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NADH 10mg 60 vcaps, Alzheimers, ATP, mitochondria, energy
NADH 10mg 60 vcaps, Alzheimers, ATP, mitochondria, energy
0 Units in Stock
Manufactured by: Now Foods
Buy from www.iherb.com
Energy, Alertness, Joints, Alzheimers, Glaucoma
Cellular Energy Production
Enhanced ATP production
Improved mitochondrial function
Protects body and brain
Supports Mental Alertness
May improve joint pain
May relieve anxiety
May halt cognitive decline in Alzheimers
With 200 mg D-Ribose
GMP Quality Assured
A Dietary Supplement
NADH (Reduced Nicotinamide Adenine Dinucleotide), also known as Co-Enzyme E1, is the metabolically active form of Vitamin B3 (Niacin).
NADH is the reduced form and NAD+ is the oxidising form, and one converts to the other as required in various enzyme reactions in the body. NAD+ combines with a hydrogen atom and 2 electrons to become NADH, essential for the production of cellular energy (ATP) from glucose and fat. Therefore, the more NADH a cell has available, the more energy it has available to operate with optimal efficiency. NADH is also directly involved in neurotransmitter production, the maintenance of genetic integrity, and the support of proper immune system function. Although NADH is normally unstable outside of the body, Panmol NADH uses a patented process to naturally preserve its effectiveness, thereby delivering stable and bioavailable NADH to the body.
Seniors have only half the natural NADH compared to the young, and extra NADH will increase the mitochondria in each cell of the body, slowing or reversing the ageing process.
In one study, patients with advanced Parkinsons disease were given NADH by IV infusions. Astoundingly, of the 480 patients who were unable to get out of their chairs, 85% could do so within two hours after their NADH IV!
In another study, Olympic cyclists were given NADH for one month, and showed an average increase in muscular energy of 7%!
Another double-blind study on Alzheimer’s patients found no evidence of progressive cognitive deterioration and significantly higher cognitive performance scores compared with those treated with placebo. Subjects took 10mg daily for six months.
As a dietary supplement, take 1 capsule daily on an empty stomach. Take with one glass of water, 30 minutes before a meal.
1 Veg Capsule
Amount Per Serving
% Daily Value
NADH (Panmol-NADH) (Reduced ß-Nicotinamide Adenine Dinucleotide)
D-Ribose (Bioenergy Ribose)
* Daily Value not established.
Cellulose (capsule), cellulose powder, stearic acid (vegetable source), silica, magnesium stearate (vegetable source) and beeswax.
Not manufactured with wheat, gluten, soy, milk, egg, fish, shellfish or tree nut ingredients. Produced in a GMP facility that processes other ingredients containing these allergens
Do not eat freshness packet. Keep in bottle.
Store in a cool, dry place after opening.
For adults only. Consult physician if pregnant/nursing, taking medication, or have a medical condition.
Keep out of reach of children
Because of constraints imposed on all by the TGA (Australian Therapeutic Goods Administration), LeanMachine does not claim that any supplement will diagnose, prevent, treat or cure any disease, sickness, condition or ailment.
Any advice or information given here is of a general nature and everyone should discuss their individual nutritional needs with their health professional.
LeanMachine cannot take responsibility for any undesirable outcome from using any product, as we have no control over individual health, use, mis-use, sensitivity or suitability.
Declining NAD+ Induces a Pseudohypoxic State Disrupting Nuclear-Mitochondrial Communication during Aging (2013) -
- this article (more technical/scientific) was the catalyst for the NAD+ research momentum which presently exists
NAD - Long-Term Administration of Nicotinamide Mononucleotide Mitigates Age-Associated Physiological Decline in Mice (2016)
- the infographic on the cover page pretty much sums up the effects in humans too
NAD - Short-term administration of Nicotinamide Mononucleotide preserves cardiac mitochondrial homeostasis and prevents heart failure (2017)
NAD - Nicotinamide mononucleotide supplementation reverses vascular dysfunction and oxidative stress with aging in mice (2016)
NAD+ repletion improves mitochondrial and stem cell function and enhances life span in mice (2016)
NAD - Loss of NAD Homeostasis Leads to Progressive and Reversible Degeneration of Skeletal Muscle (2016)
NAD - Nicotinamide mononucleotide improves energy activity and survival rate in an in vitro model of Parkinson's disease (2014)
NAD - Nicotinamide Mononucleotide, a Key NAD+ Intermediate, Treats the Pathophysiology of Diet- and Age-Induced Diabetes in Mice (2011)
NAD+ in aging, metabolism, and neurodegeneration (2015)
NAD - Exogenous NAD+ administration significantly protects against myocardial ischemia-reperfusion injury in rat model (2016)
NAD - Prevention of Traumatic Brain Injury-Induced Neuron Death by Intranasal Delivery of Nicotinamide Adenine Dinucleotide (2012)
NAD+ controls neural stem cell fate in the aging brain (2014)
NAD+ Deficiency in Age-Related Mitochondrial Dysfunction (2014)
NAD - The NAD+ Precursor Nicotinamide Riboside Enhances Oxidative Metabolism and Protects against High-Fat Diet-Induced Obesity (2012)
NAD - Nicotinamide mononucleotide inhibits post-ischemic NAD+ degradation and dramatically ameliorates brain damage following global cerebral ischemia (2016)
NAD - Effect of nicotinamide mononucleotide on brain mitochondrial respiratory deficits in an Alzheimer’s disease-relevant murine model (2015)
NAD - Nicotinamide Mononucleotide, an Intermediate of NAD+ Synthesis, Protects the Heart from Ischemia and Reperfusion (2014)
NAD - Restoration of Mitochondrial NAD+ Levels Delays Stem Cell Senescence and Facilitates Reprogramming of Aged Somatic Cells (2016)
NAD - Nicotinamide Mononucleotide, an NAD+ Precursor, Rescues Age-Associated Susceptibility to Acute Kidney Injury [was 'AKI'] in a Sirtuin 1–Dependent Manner (2017)
NAD - NAD replenishment with nicotinamide mononucleotide protects blood–brain barrier integrity and attenuates delayed tissue plasminogen activator-induced haemorrhagic transformation after cerebral ischaemia (2017)
NAD - Nicotinamide mononucleotide inhibits JNK activation to reverse Alzheimer disease 
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