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How Aluminum Damages Your Brain


Reproduced from original article:
https://articles.mercola.com/sites/articles/archive/2020/02/19/aluminum-and-brain-damage.aspx
Analysis by Dr. Joseph Mercola       –  February 19, 2020

aluminum and brain damage

STORY AT-A-GLANCE

  • Research has found a strong link between aluminum exposure and Alzheimer’s disease. Patients with a genetic mutation that predisposes them to early onset of Alzheimer’s and more aggressive disease have universally high aluminum content in their brains
  • According to a British researcher, without aluminum in the brain, Alzheimer’s does not develop
  • When aluminum was first approved for use in vaccines, it was approved based on its efficacy. It was never actually tested for safety. It was simply assumed to be safe
  • Aluminum has been shown to cause mitochondrial dysfunction and depletion of adenine-triphosphate (ATP), which sets the stage for virtually any chronic disease. Aluminum salts can increase levels of glial activation, inflammatory cytokines and amyloid precursor protein within the brain
  • Recent research found the U.S. Centers for Disease Control and Prevention’s vaccine schedule — when adjusted for bodyweight — exposes children to a level of aluminum that is 15.9 times higher than the recommended “safe” level

For years, I’ve warned that aluminum is a serious neurotoxic hazard involved in rising rates of autism and Alzheimer’s disease (AD). I’ve also warned that vaccines are a significant source of such exposure, and may be one of the worst, since by injecting it, the aluminum bypasses your body’s natural filtering and detoxification systems.

My comments above were one of the reasons the self-appointed global arbiter of fake news, NewsGuard, refused to give us “green” status as a site that follows “basic standards of accuracy and accountability.” In other words, our reporting of aluminum hazards was deemed “fake news.”

Not only were my earlier reports based on published science, but now we have yet another study,1 published in the Journal of Alzheimer’s Disease, strongly linking aluminum exposure to AD. As reported by SciTech Daily:2

“Researchers found significant amounts of aluminum content in brain tissue from donors with familial AD. The study also found a high degree of co-location with the amyloid-beta protein, which leads to early onset of the disease.

‘This is the second study confirming significantly high brain accumulation in familial Alzheimer’s disease, but it is the first to demonstrate an unequivocal association between the location of aluminum and amyloid-beta in the disease.

It shows that aluminum and amyloid-beta are intimately woven in the neuropathology,’ explained lead investigator Christopher Exley, PhD, Birchall Centre, Lennard-Jones Laboratories, Keele University, Staffordshire, UK.”

The Association Between Aluminum and Amyloid-Beta

To gain a better understanding of the link between aluminum exposure and beta-amyloid generation, the researchers examined the brain tissue of donors diagnosed with familial Alzheimer’s disease who also had a specific gene mutation known to increase levels of amyloid-beta, leading to early onset and more aggressive disease.

Aluminum levels were compared to controls with no neurological disease diagnosis. They found striking differences between these two groups. Donors with the genetic mutation had universally high aluminum content.

While all samples had some level of aluminum, 42% of the samples from those with familial Alzheimer’s had “pathologically significant” aluminum levels, and the aluminum was primarily co-located with amyloid beta plaques. As reported by SciTech Daily:3

“The results strongly suggest that genetic predispositions known to increase amyloid-beta in brain tissue also predispose individuals to accumulate and retain aluminum in brain tissue …

‘One could envisage increased amyloid-beta in brain tissue as a response to high levels of aluminum content, or that aluminum fosters the accumulation of amyloid-beta,’ said Dr. Exley.

‘Either way, the new research confirms my resolve that within the normal lifespan of humans, there would not be any AD if there were no aluminum in the brain tissue. No aluminum, no AD.'”

Aluminum Adjuvants Have Never Been Tested for Safety

Exley’s conclusion deserves repeating: “No aluminum, no AD.” Without aluminum, Alzheimer’s doesn’t develop. That’s not fake news. This research provides conclusive evidence for concern, which means it would be foolish in the extreme to pretend that injecting infants and young children with aluminum-containing vaccines is harmless.

As revealed in my 2015 interview with Dr. Lucija Tomljenovic, featured in “How Vaccine Adjuvants Affect Your Brain,” when aluminum was first approved for use in vaccines, some 95 years ago, it was approved based on its efficacy. It was never actually tested for safety.

Even the total allowable limit was based on efficacy data, not safety data. They simply assumed it was safe. As noted by Tomljenovic in that interview:

“A document4 from 2002 from the U.S. Food and Drug Administration (FDA) … discussing the assessment of vaccine ingredients … and testing specifically in animal models … stated that the routine toxicity studies in animals with vaccine ingredients have not been conducted because it was assumed that these ingredients are safe.

When I read that I was kind of pulling my hairs out [thinking] ‘So, this is your indisputable evidence of safety?’ These documents never made it to mainstream media. It’s just a lie perpetuated over and over again; that we’ve been using these things for over nine decades and it’s been proven safe. No, it’s been ASSUMED safe.”

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Industry Propaganda and Political Interference

The propaganda responsible for hiding the dangers of aluminum was addressed in a 2014 review article5 in the journal Frontiers of Neurology. In it, Exley (who also co-authored the featured Journal of Alzheimer’s Disease study above) wrote:6

“The aluminum industry is a pillar of the developed and developing world and irrespective of the tyranny of human exposure to aluminum it cannot be challenged without significant consequences for businesses, economies, and governments …

There has been and there continues to be systematic attempts by the aluminum industry to suppress research on aluminum and human health.

While independent research in this field is prevented the questions concerning human toxicity remain unanswered. Lack of required research does not equate to lack of biological effect or safety … 

Herein, I will make the case that it is inevitable both today and in the future that an individual’s exposure to aluminum is impacting upon their health and is already contributing to, if not causing, chronic diseases such as Alzheimer’s disease.”

Exley points out that one of the most significant factors driving complacency about aluminum exposure is the aluminum industry’s insistence that, since it’s everywhere and found in virtually everybody,7 it must be harmless if not essential — we just haven’t figured out how it benefits us yet. However, no beneficial role of aluminum has ever been elucidated, and its presence is in no way evidence of benefit.

Why Aluminum Toxicity Flies Under the Radar

Exley also notes that aluminum is rarely acutely toxic, which adds to the complacency problem. Problems only arise once a certain threshold is reached, and even then, its role in disease is rarely if ever investigated.

Yet another factor that helps hide the influence of aluminum in disease is the fact that it acts on many different pathways and acts as a substitute for essential minerals, so aluminum toxicity doesn’t have one specific hallmark.

“The potential for aluminum to interact with and to influence so many biochemical pathways means that the symptoms of its toxicity could be deficiency or sufficiency, agonistic or protagonistic, and any combination of these and other physiology-based events,” Exley writes, adding:8

“For aluminum to play a significant role in any disease-related event some degree of toxicity threshold must have been achieved. Essentially, the rate of delivery of Al3+(aq) to target ligands must be sufficient to overcome the inherent robustness of systems that are under attack.

In achieving this threshold either aluminum must accumulate over time within a particular compartment or possibly the administration of a single dose of aluminum could achieve such a threshold instantaneously.

The latter is probably more unusual in human being’s everyday exposure to aluminum except, for example, where aluminum is administered as an adjuvant in vaccination and allergy immunotherapy.”

Importantly, aluminum has the ability to cross the blood-brain-barrier, so any aluminum in the blood can be transported into the brain. “Indeed, aluminum is known to increase the leakiness of epithelial and endothelial barriers and in doing so could concomitantly increase the passage of aluminum from the blood to the brain,” Exley writes.9

Biological Effects of Aluminum

Exley also points out aluminum can damage your brain function by:

  • Adversely influencing neuronal function and survival
  • Potentiating damaging redox activity
  • Disrupting intracellular calcium signaling that systematically wears down cellular defenses
  • Worsening the adverse effects of other heavy metals
  • Influencing gene expression

A 2010 paper10 also pointed out that aluminum salts “can increase levels of glial activation, inflammatory cytokines and amyloid precursor protein within the brain,” and that “Both normal brain aging and to a greater extent, Alzheimer’s disease are associated with elevated basal levels of markers for inflammation.”

Similarly, a 2018 paper11 in the Journal of Research in Medical Sciences cites research showing aluminum affects:

Axonal transport Neurotransmitter synthesis
Synaptic transmission Phosphorylation or de-phosphorylation of proteins
Protein degradation Gene expression
Peroxidation Inflammatory responses

When it comes to altering gene expression, aluminum has been shown to do this via many different routes and mechanisms, including by:12

Binding to histone-DNA complex Inducing conformational changes of chromatin
Inducing topological changes of DNA Decreasing expression of neurofilament
Decreasing expression of tubulin Altering expression of neurofilament genes
Altering expression of amyloid precursor protein Altering expression of neuron-specific enolase
Decreasing expression of transferrin receptor Altering expression of RNA polymerase I
Altering expression of oxidative stress marker genes such as SOD1 and glutathione reductase Altering expression of beta-APP secretase

Importantly, as noted in the Journal of Research in Medical Sciences, aluminum has been shown to “cause mitochondrial dysfunction and depletion of adenine-triphosphate (ATP),”13 which sets the stage for virtually any chronic disease, not just neurodegenerative diseases.

Vaccine Schedule Overexposes Infants to Aluminum

In December 2019, The Highwire reported14 the findings of a study15 published in the Journal of Trace Elements in Medicine and Biology, which found the U.S. Centers for Disease Control and Prevention’s childhood vaccine schedule — when adjusted for bodyweight — exposes children to a level of aluminum that is 15.9 times higher than the recommended “safe” level.

The researchers point out that previous efforts to assess the aluminum burden created by vaccines were based on “whole-body clearance rates estimated from a study involving a single human subject.”

What’s more, they used an aluminum citrate solution that is not used in vaccines, which may affect the excretion rate. Importantly, infants also have immature renal function, which will inhibit their ability to filter and excrete toxins in the first place.

Other studies16 have used orally ingested aluminum to assess and defend safety limits for aluminum in vaccines. This is clearly an unwise comparison, as only 0.1% of orally ingested aluminum is absorbed and made bioavailable from the gastrointestinal tract.17,18

In the Journal of Trace Elements in Medicine and Biology study,19 the researchers used several different models in an effort to estimate the expected acute and long-term whole-body accumulation of aluminum in children following one of the three possible vaccine schedules:

  1. The CDC’s childhood vaccine schedule as of 2019
  2. The CDC’s vaccine schedule modified to use low dose aluminum DTaP and aluminum-free Hib vaccines
  3. Dr. Paul Thomas’ “vaccine-friendly plan,”20,21 which recommends giving only one aluminum-containing vaccine per visit (max two) and delaying certain vaccinations

The CDC’s standard schedule resulted in the greatest expected aluminum burden in all model assumptions, while Thomas’ schedule resulted in the lowest. According to the authors:22

Medically, proper organ, cellular and body aluminum detoxification appears to be of ever-increasing importance: Aluminum has been found in the brains of patients with Parkinson’s Disease, Alzheimer’s disease, epilepsy, and autism.

Evidence is growing that a host of chronic illnesses of unknown cause that are difficult to diagnose such as PANDAS/PANS, chronic fatigue syndrome may at least in part be due to vaccine aluminum intolerance.

Aluminum compounds occur naturally in the environment and in food, but very little ingested aluminum is absorbed through the intestines. Total aluminum exposure is affected by the aluminum amount in individual vaccines and the timing of repeated vaccinations in the first two years of life.

Dórea and Marques compared the expected levels of aluminum uptake into the body from intravenous and oral intake and concluded that human infants have higher exposure to aluminum from vaccination than from food, water, and formula.

Our calculations confirm that for the CDC schedule, infants up to six months of life receive most of their metabolically available aluminum from vaccines.

It should be expected that most aluminum retained in the body of infants comes from vaccinations combined with the levels of exposure from other exposures to manifest health risks from total exposure, making the timing and total aluminum content of different vaccine schedules an important consideration.”

CDC Vaccine Schedule Exceeds Aluminum Limit for Adults

As noted in the Journal of Trace Elements in Medicine and Biology study,23 the “safety” limit for aluminum is not weight dependent. The maximum safe limit is based on an adult, and the same limit is transposed to infants weighing a fraction of that.

Importantly, this study found that when multiple aluminum-containing vaccines are given together, as per the CDC schedule, the total aluminum dose ends up exceeding even the assumed safety limit for an adult.

“Adjusting the safe dose limit based on a child’s weight at these ages therefore results in doses that far exceed the estimated safe limit of acute toxicity,” the authors warn,24 adding that “on all days of injection the safe limit for a child is exceeded for all three schedules; this points to acute toxicity …

The CDC schedule has the largest violation at 15.9 times the recommended safe level. This occurs at 2 months, when four recommended vaccinations containing aluminum are simultaneously administered.

In addition, modeling the time to clear aluminum from the body using Priest’s equation estimates that for this schedule a child will be over the safe level of aluminum in the body for 149 days from birth to 7 months, constituting about 70 % of days in this period. This points to chronic toxicity …

The modified CDC schedule assumes the same vaccinations at the same times as the CDC schedule, but like the Vaccine Friendly Plan it assumes a lower dose aluminum DTap vaccine, and also combines the ActHib (containing no Al) with low aluminum DTap or PVC13 so that the aluminum adjuvant in the aluminum containing vaccine (ACV) activates an immune response for the ActHib vaccine.

This drops the maximum level of exposure to about 60 % of the original CDC plan with (from 15.9 to 9.3) and drops days above the estimated safe limit in the first 7 months from 70 % of days to 26 % and in the first 2 years from 24 % of days to 8 %.

The Vaccine Friendly Plan schedule skips some vaccinations in the first two years (like HepB) and avoids giving more than two vaccinations containing aluminum together.

The VFP thus further limits maximum exposure to approximately 25 % of the original CDC schedule (from 15.9 to 4.2) and drops days above the estimate limit in the first seven months from 70 % of days to 5 % and in the first two years from 24 % of days to 2 %.”

Aluminum Is a Proven Neurotoxin

The health hazards of aluminum are also addressed in a 2017 scientific review25 published in the German journal, Deutsches Ärzteblatt International, which also reviews the threshold values associated with various types of exposure.

“Aluminum’s neurotoxic effects in humans and its embryotoxic effects in animal models have been proven,” the paper states, adding that while the acute toxicity of ingested aluminum is low, long-term exposure and buildup is associated with neurotoxic effects, resulting in disorientation, memory impairment and dementia. As noted in this paper:26

“In addition to inducing oxidative stress and binding to negatively charged membrane structures in neurons, aluminum is able to modify hippocampal calcium signal pathways that are crucial to neuronal plasticity and, hence, to memory. Cholinergic neurons are particularly susceptible to aluminum neurotoxicity, which affect synthesis of the neurotransmitter acetylcholine.”

Aluminum as a risk factor for neurological disorders is also detailed in a 2018 paper27 in the Journal of Research in Medical Sciences. Here, the authors again note that “it is widely accepted that [aluminum] is a recognized neurotoxin, which could cause neurodegeneration.” They also point out that aluminum “affects more than 200 important biological reactions and causes negative effects on [the] central nervous system.”

Aluminum Detected in Organs a Year After Vaccination

A 2013 study28 shed important light on the vaccine adjuvant alum, a “nanocrystalline compound” that has been shown to spontaneously form “micron/submicron-sized agglomerates.” According to this paper:

“Alum is occasionally detected within monocyte-lineage cells long after immunization in presumably susceptible individuals with systemic/neurologic manifestations or autoimmune (inflammatory) syndrome induced by adjuvants (ASIA) …

Intramuscular injection of alum-containing vaccine was associated with the appearance of aluminum deposits in distant organs, such as spleen and brain where they were still detected one year after injection …

Particles linearly accumulated in the brain up to the six-month endpoint; they were first found in perivascular CD11b+ cells and then in microglia and other neural cells … Cerebral translocation was not observed after direct intravenous injection, but significantly increased in mice with chronically altered blood-brain-barrier …

Continuously escalating doses of this poorly biodegradable adjuvant in the population may become insidiously unsafe, especially in the case of overimmunization or immature/altered blood brain barrier or high constitutive CCL-2 production.”

Clearly, Alzheimer’s and autism are not caused by a single factor. Your diet and lifestyle play significant roles, as do other toxic exposures. Still, aluminum appears to be a significant concern that cannot be overlooked, especially where vaccines are concerned. Can we really justify loading infants up with aluminum at doses that are toxic even to an adult?

To learn more about the factors that raise your risk for Alzheimer’s and recommended prevention strategies, see “How Excess Iron Raises Your Risk for Alzheimer’s,” “Trans Fats Linked to Increased Risk for Alzheimer’s,” and “Lifestyle Factors Linked to Alzheimer’s.”

– Sources and References

The Importance of Lutein for Eye and Brain Health


Reproduced from original article:
https://articles.mercola.com/sites/articles/archive/2020/02/10/lutein-eye-health.aspx
Analysis by Dr. Joseph Mercola     

February 10, 2020

STORY AT-A-GLANCE

  • Lutein is well-known for its vision-enhancing properties. Research also suggests it has neuroprotective qualities, improving memory and boosting intelligence
  • There’s an inverse association between lutein levels in the eyes and age-related macular degeneration (AMD), the primary cause of blindness in the elderly, as well as cataracts
  • Lutein has also been shown to prevent cell death caused by retinal detachment when administered in a timely manner, and can help improve night vision
  • Lutein may help prevent neurodegenerative diseases by preventing DNA damage, the depletion of BDNF and the degradation of a synaptic vesicle protein involved in Alzheimer’s and Parkinson’s. It’s also been shown to help maintain the integrity of your brain’s white matter
  • Your body cannot manufacture lutein, so you need to get it from your diet. Lutein is a carotenoid found in egg yolks, avocados, cruciferous vegetables and dark, leafy greens, especially kale and spinach

Lutein — a carotenoid found in egg yolks, avocados, cruciferous vegetables and dark, leafy greens — is well-known for its vision-enhancing properties.1 Research also suggests it has neuroprotective qualities,2 improving memory3 and boosting intelligence.4 As reported in the Senior Resource Guide:5

“Lutein belongs in the subclass of xanthophylls, which have polar molecular structures that possess unique membrane properties, such as fluidity, communication between brain cells, ion exchange, diffusion of oxygen, membrane stability, and the prevention of oxidation and inflammation.

Like other xanthophylls, lutein accumulates in neural tissue. In fact, lutein accounts for most of the carotenoid accumulation in the human brain, and this high concentration of lutein in the brain may indicate its neuroprotective value.

Lutein accumulates in the brain and embeds in cell membranes. There, lutein protects the neuronal structure and function of brain cells. While lutein is distributed in the gray matter of the brain, researchers have detected its presence in the temporal cortex, the prefrontal cortex, and the hippocampus. Lutein accumulates in the brain over a person’s lifespan and may therefore provide lifelong benefits to brain health.”

Your body cannot manufacture lutein, so you need to get it from your diet. Unfortunately, many do not get enough lutein simply because they don’t eat enough vegetables and/or eggs.

How Lutein Protects Your Vision

As its name implies, lutein is found in high concentrations in your macula lutea,6 the small central part of your retina responsible for detailed central vision. It’s also found in your macular pigment (it’s responsible for the yellow hue of the macula7) and the lens of your eye.

Epidemiological research has found an inverse association between lutein and zeaxanthin levels in the eyes and age-related macular degeneration (AMD), the primary cause of blindness in the elderly, and cataracts.8,9

As explained in the 2013 paper,10 “The Role of Lutein in Eye-Related Disease,” lutein and zeaxanthin (another carotenoid present in high concentration in the eyes) help prevent age-related eye degeneration by ameliorating the damage caused by blue light and oxygen free radicals. Blue light is responsible for a majority of the damage to the eye, and lutein selectively absorbs blue light.

According to this paper, “Dietary concentrations between 6 and 20 mg per day of lutein have been associated with a reduced risk of ocular disorders such as cataracts and age-related macular degeneration.”

A more recent scientific review,11 published in 2019 in PLOS ONE, found people who took 10 mg or 20 mg of lutein per day had greater macular pigment optical density (MPOD), and better visual acuity and contrast sensitivity than those who did not supplement.

Macular pigment, the yellow pigmented area found in the center of your retina, acts as “internal sunglasses,” protecting your macula from harmful blue light. The denser your macular pigment, the lower your risk of developing AMD. Greater MPOD is also associated with better visual performance overall.12

According to the authors of the 2019 PLOS ONE review, “The available evidence suggests that dietary lutein may be beneficial to AMD patients and the higher dose could make MPOD increase in a shorter time.” Lutein has also been shown to prevent cell death caused by retinal detachment, when administered in a timely manner,13 and can help improve night vision.14,15

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Lutein’s Role in Brain Health and Cognition

More recent research has also found lutein plays an important role in brain health, and may even help prevent neurodegenerative diseases.16 As noted in the 2012 paper,17 “Neuroprotective Effects of Lutein in the Retina,” lutein benefits your brain and cognition by preventing:

  • DNA damage
  • Depletion of brain-derived neurotrophic factor (BDNF), which is important for brain health
  • Degradation of synaptophysin, a synaptic vesicle protein involved in neurodegenerative diseases such as Alzheimer’s and Parkinson’s disease

Lutein and zeaxanthin have also been shown to help maintain the integrity of your brain’s white matter,18 and this too can lower your risk of age-related decline.

Lutein Benefits Both the Old and the Young

In one 2017 study,19,20,21 which involved 60 adults between the ages of 25 and 45, those with higher levels of lutein in middle-age had more youthful neural responses than those with lower levels.

Carotenoid status was assessed by measuring MPOD, which is also highly correlated with the lutein status in your brain. Most studies have focused on the effects of diet after cognitive decline has already set in.

Here, they wanted to evaluate whether lutein might have a preventive effect, as the process of cognitive decline has been shown to begin far earlier than typically expected. According to the researchers, you can start seeing cognitive deterioration as early as your 30s.

Indeed, the results suggest your diet, and in this case lutein-rich foods, does help keep your brain young. As noted by co-author Naiman Khan, professor of kinesiology and community health at the University of Illinois:22

“Now there’s an additional reason to eat nutrient-rich foods such as green leafy vegetables, eggs and avocados. We know these foods are related to other health benefits, but these data indicate that there may be cognitive benefits as well.”

Lutein has also been shown to benefit younger people. In one such study,23 healthy 18- to 30-year-olds exhibited better brain function after taking lutein and zeaxanthin supplements for a year. Significant improvements were seen spatial memory, reasoning ability and complex attention.

Cognitive Benefits Associated With Higher Lutein Levels

Several other studies support these findings. For example, in one University of Georgia study,24 older adults (mean age 72) taking 10 mg of lutein and 2 mg of zeaxanthin daily for one year were able to maintain their brain function, whereas the placebo group’s verbal learning ability and word recall deteriorated over the course of the study.

The mechanism thought to be responsible for this protective effect was enhanced blood flow through the brain. A number of other studies looking at lutein’s influence on cognition have shown that higher lutein and zeaxanthin levels in the macula are associated with improved:25,26,27

Verbal learning and fluency

Word recall

Executive function such as sorting and prioritizing information and the ability to take action

“Crystallized intelligence”28 — the ability to use learned knowledge and experience (opposed to the ability to logically reason your way through a new situation or problem, which is known as “fluid intelligence”29)

Relational memory performance30,31 — the ability to remember a person’s name when seeing their face, or to retell a story

Other Health Benefits of Lutein

Lutein has also been found to promote health in other ways, beside optimizing vision and cognition. For example, studies have found:

Diets rich in the carotenoids beta-carotene, lutein and lycopene resulted in greater resistance against oxidation of low-density lipoprotein (LDL) cholesterol. Higher plasma concentration of carotenoids was also associated with lower DNA damage.32
Lutein and zeaxanthin in combination with vitamin E appears to improve lung function and respiratory health.33
Plasma levels of antioxidants such as lutein, zeaxanthin, vitamin E, beta-cryptoxanthin, lycopene, alpha-carotene and beta-carotene are inversely correlated with congestive heart failure severity.34
Plasma carotenoid levels are also inversely correlated with prostate cancer.35
Lutein and lycopene enhance eye health by reducing your risk for oxidative stress-induced loss of retinal pigment epithelial (RPE) cells, and inhibiting the cell growth in undifferentiated RPE cells.36
Lutein has been shown to induce autophagy and may help shield your body against certain stresses.37
Lutein and zeaxanthin have also been shown to increase bone density in young, healthy adults.38 Lutein appears to stimulate bone mineralization and formation by suppressing bone resorption.

Lutein-Rich Foods

Lutein is primarily found in green leafy vegetables, with kale and spinach topping the list of lutein-rich foods. You’ll also find it in orange- and yellow-colored fruits and vegetables. The word lutein actually comes from the Latin word “luteus,” which means “yellow.”

As a general rule, anywhere from 15% 47% of the total carotenoid content in dark green leafy vegetables is lutein.39 Following is a list of foods that are particularly rich in lutein.40,41,42 Most of these also contain zeaxanthin, albeit in lesser quantities than lutein.

  • Egg yolks
  • Kale and spinach
  • Avocado
  • Broccoli
  • Green, red and yellow peppers

Ideally, you’ll want to buy the whole food and consume these foods as close to raw as possible, as the lutein (and other carotenoids such as zeaxanthin) are easily damaged by heat. Accessory micronutrients in the foods that enhance their action also tend to get easily damaged.

While there’s no recommended daily intake for lutein or zeaxanthin, studies have found health benefits for lutein at a dose of 10 milligrams (mg) per day and at 2 mg/day for zeaxanthin.

How to Optimize Lutein Absorption

Lutein and other carotenoids are fat-soluble, so to optimize absorption, be sure to add a little bit of healthy fat to your meal. For example, research43,44 shows that adding a couple of eggs — which contain both lutein and healthy fats — to your salad can increase the carotenoid absorption from the whole meal as much as ninefold.

Ideally, opt for organically-raised, free-range pastured eggs. Not only do they tend to have a better nutritional profile, by opting for pastured eggs you’ll also avoid pesticide exposure and genetically modified organisms. I raise six chickens and typically eat five eggs a day — the yolks raw in my smoothie and the whites cooked with my meat.

The vast majority of commercially available eggs come from concentrated animal feeding operations (CAFOs), where the hens are not permitted to forage on pasture. Instead, they’re typically fed a diet of corn and soy, the vast majority of which are genetically engineered. CAFO eggs are also far more prone to cause foodborne illness caused by salmonella contamination

If you live in an urban area, visiting a local health food store is typically the quickest route to finding high-quality local egg sources. Your local farmers market is another source for fresh free-range eggs. Cornucopia.org also offers a helpful organic egg scorecard45 that rates egg manufacturers based on 22 criteria that are important for organic consumers.

You can often tell the eggs are free-range by the color of the egg yolk. Foraged hens produce eggs with bright orange yolks, indicative of higher amounts of lutein and zeaxanthin. Another way to boost absorption of lutein from your vegetables is to add some raw organic butter or healthy oil such as olive or coconut oil to your salad.

Eat Right to Avoid AMD

To protect your vision over the long haul, aside from making sure you’re eating plenty of lutein-rich foods, you’ll also want to avoid a high-glycemic diet. As reported by Tufts University in 2017:46

“Sheldon Rowan, a scientist in the Laboratory for Nutrition and Vision Research at the Human Nutrition Research Center on Aging at Tufts, said there are plenty of indications that the types of carbohydrates we eat play a role in the development of AMD.

People who eat lots of simple carbohydrates, like those in white bread and sweetened beverages, are more likely to get the disease. This could be because simple carbs break down rapidly during digestion, creating a spike in blood sugar that can lead to widespread inflammation, a condition linked to AMD … If … blood glucose stays low over a long period of time, Rowan said, it can lower incidence of AMD.

To understand why, Rowan tested the two diets on laboratory mice. Over the course of a year, he fed one group of mice ‘high-glycemic’ foods — ones with lots of simple starches. A second group got a ‘low-glycemic’ diet, rich in complex carbs, but otherwise identical in calories and nutrients. In a third group, Rowan switched the mice’s diet from high- to low-glycemic foods halfway through the study.

Sure enough, mice with the low-glycemic diet did not develop AMD, while mice fed the high-glycemic diet almost all came down with the disease … In the mice that switched diets, though, Rowan saw something completely unexpected. Not only did they avoid AMD, but the existing damage to their retinas was reversed.

‘No one had ever seen that before,’ Rowan said of the findings, which were reported in Proceedings of the National Academy of Sciences.47 ‘The most common form of AMD doesn’t really have a treatment right now — but this suggests that just changing to a healthier eating pattern could have a huge impact.’”

Further exploration revealed that high-glycemic diets led to higher levels of advanced glycation end products (AGEs), which Rowan points out are “toxic end products of sugars” that “can damage the proteins and lipids that a cell needs to function.”

In the retina, the damaged proteins accumulate forming drusen, yellow deposits that damage your retinal cells.48 Elevated insulin levels also affect the development of your eyeball, making it abnormally long, thereby causing near-sightedness.49

Following my nutrition plan will help normalize your insulin level by reducing, or eliminating, excess sugar and processed grains from your diet. To learn more about which foods can help safeguard your vision, please see my previous articles, “Eat Right to Protect Your Eyesight,” and “The Best Foods for Healthy Eyes.”

– Sources and References

Coconut Oil Improves Brain Function in Alzheimer’s Patients

© 11th January 2020 GreenMedInfo LLC. This work is reproduced and distributed with the permission of GreenMedInfo LLC. Want to learn more from GreenMedInfo? Sign up for the newsletter here www.greenmedinfo.com/greenmed/newsletter

Reproduced from original article:
www.greenmedinfo.com/blog/coconut-oil-improves-brain-function-alzheimer-s-patients

Alzheimer’s disease is a devastating degenerative brain condition that affects millions of people in the U.S. While pharmaceutical treatments have long lists of side effects, there is a natural food-based intervention that has proven effective in improving key brain functions. The best part is, it’s probably in your food pantry right now

If you’re a regular reader of GreenMedInfo.com, you’re likely to have seen numerous articles detailing the dozens of healthy uses for coconut oil that are backed by science. From balancing blood sugar[i] and hormones[ii] to healing burns[iii] and ulcers,[iv] it seems there is hardly an ailment that is not soothed or supported by adding this nutrition-dense fat to your diet.

Coconut Oil: The Brain’s Preferred Fuel?

In 2018, researchers added to the knowledge base with confirmation of coconut oil’s usefulness as a brain-boosting superfood. The pilot study,[v] published in July 2018 in the Journal of Alzheimer’s Disease, has shown that a Mediterranean diet, rich in coconut oil, improves the main cognitive functions in patients with Alzheimer’s disease (AD).

Conducted by a multidisciplinary team of researchers from the Catholic University of Valencia, Spain, the aim of the study was to detect changes in key cognitive functions of patients with AD after following a traditional Mediterranean diet boosted by therapeutic doses of coconut oil.

Study methods were prospective, longitudinal, qualitative and analytic, meaning participants’ health and behaviors were studied across time to observe unknown and unpredicted changes in outcomes. Inclusion criteria were diagnosed AD patients, aged 65 to 85 years old, who were institutionalized in the Alzheimer’s Family Association of Valencia (AFAV).

A representative sample size of 44 participants was ultimately selected from the original pool of 458 AFAV patients, with criteria excluding patients who were diagnosed with other types of degenerative cognitive disorder or verbal disability that prevented them from answering test questions, and excluding patients with any metabolic chronic disease or who had been treated with drugs such as antidepressants, antipsychotics or hypnotic drugs, which could alter cognitive functions.

The 44 participants were randomly divided into two homogenous groups comprised of 22 patients each: an experimental group receiving coconut oil supplementation and a control group that did not receive coconut oil. Both groups followed an isocaloric Mediterranean diet that was shown in previous studies to be associated with a decrease in cognitive impairment in AD patients.

In the Mediterranean diet implemented in this study, proteins accounted for 15% of total calories, carbohydrates for 55% and lipids for 30% of overall energy intake. Calorie intake was the same for all participants, taking into account that in the experimental group, lipids were reduced so that by adding the coconut oil supplement, the daily lipid amount for all study participants was the same. The dietary intervention was conducted over a period of 21 days.

Cognitive changes in participants were measured by the same institutional psychologist, blind to study protocols, who conducted the “7-Minute Screen,” an assessment that measures “temporal orientation, visuospatial and visuoconstructive abilities.” Visuoconstructive disabilities are represented by difficulty doing math, driving and writing, among other common daily tasks. Patients were assessed the day before dietary therapy and the day after therapy throughout the 21-day intervention.

Alzheimer’s: Most Prevalent Brain Disorder

According to researchers, “Alzheimer’s disease (AD) is the most prevalent neurodegenerative disorder, and new therapies are needed.” This study was a method of proving their hypothesis that coconut oil can be a beneficial source of ketone bodies, an alternative brain fuel to glucose for AD patients whose hypometabolism, or poor glucose utilization, is a factor in their disease.

In addition to serving as a fuel source for brain metabolism, ketone bodies, in adequate doses, regulate glutamate release in the synaptic cleft, the space between neurons that transmits impulses.[vi] Glutamate is a neurotransmitter that is involved in most aspects of normal brain function.[vii]

Researchers stated that gender is a significant factor in AD, with women more commonly affected than men.

The trial groups had 75% female and 25% male patients, reflecting the same percentages of gender distribution as the disease itself.[viii] In the published paper, the scientists noted the insidious onset of Alzheimer’s disease, which initially becomes apparent through “progressive loss of episodic memory, followed by gradual impairment of declarative and non-declarative memory.

Later, loss of other main cognitive functions, such as language, executive functions, attention span, and working memory, have also been observed as well as alterations in temporal orientation, visuospatial ability, and visuoconstructive ability.”[ix]

After a baseline assessment of all participants using the 7-Minute Screen, dosing commenced consisting of 20 milliliters (mls) of coconut oil, twice daily, for a total daily dose of 40 mls. This amount of coconut oil had previously demonstrated effectiveness at improving cognitive functions over 21 days in human[x] and mice studies.[xi]

Coconut Oil Improves Information Processing and Memory in AD Patients

Results were both confirming of the researchers’ hypothesis regarding the benefits of coconut oil and encouraging for proponents of natural disease interventions:

“Taking a closer look at the changes observed in the group that received coconut oil, these changes seem to point to the fact that certain cognitive functions improved … such as temporal orientation (information processing), semantic memory and episodic memory …

[These improvements] … could be explained by the decrease in insulin resistance due to the action of ketone bodies, since memory improvement has been observed after intranasal administration of insulin in AD patients, which increases glucose metabolism.”[xii]

An important observation was made regarding the potential for brain recovery with coconut oil: “It could be deduced that not all regions of the cerebral cortex recover to the same degree.”[xiii] Regarding gender differences, researchers observed that “female patients recover more easily than male patients, which confirms our previous results, where a global cognitive improvement was shown in women.”[xiv]

They hypothesize that these results could possibly be explained by hormonal differences in sex, “but not only with respect to low estrogen levels but also … by testosterone, whose levels of production are much lower in women with AD and cause them to have higher insulin resistance.”[xv] Researchers concluded that the positive effects of coconut oil are not gender- or state-specific, however, the benefits are “more evident in women with mild-moderate state [AD].”

Final conclusions of the study were that an isocaloric, coconut oil-enriched Mediterranean diet improves cognitive functions in patients with AD, with differences according to patient sex and degree of severity of the disease.[xvi] They issued a call for further studies of this type to add to this important body of evidence.

To learn more about the health benefits of coconut oil, GreenMedInfo.com has more than 70 abstracts in the world’s most widely referenced natural health database.


References

[i] Protective and Antidiabetic Effects of Virgin Coconut Oil (Vco) on Blood Glucose Concentrations in Alloxan Induced Diabetic Rats. Nur‘azimatul Quddsyiah H. Maidin, Norhayati Ahmad. International Journal of Pharmacy and Pharmaceutical Sciences. Vol 7, Issue 10, 2015. ISSN: 0975-1491. https://pdfs.semanticscholar.org/62d6/b586d89f623b4be84ac93c828b31f1070b76.pdf

[ii] Effect of dietary saturated fatty acids on hormone-sensitive lipolysis in rat adipocytes. Awad AB, Chattopadhyay JP. J Nutr. 1986 Jun;116(6):1088-94. PMID: 3014093

[iii] Burn wound healing property of Cocos nucifera: An appraisal. Srivastava P, Durgaprasad S. Indian J Pharmacol. 2008 Aug;40(4):144-6. doi: 10.4103/0253-7613.43159. PMID: 20040946

[iv] Antiulcerogenic effects of coconut (Cocos nucifera) extract in rats. Nneli RO, Woyike OA. Phytother Res. 2008 Jul;22(7):970-2. doi: 10.1002/ptr.2318. PMID: 18521965

[v] Improvement of Main Cognitive Functions in Patients with Alzheimer’s Disease after Treatment with Coconut Oil Enriched Mediterranean Diet: A Pilot Study.  de la Rubia Ortí JE, et al. J Alzheimers Dis. 2018;65(2):577-587. doi: 10.3233/JAD-180184. PMID: 30056419

[vi] Mirriam-Webster, Medical, Synaptic-cleft, https://www.merriam-webster.com/medical/synaptic%20cleft

[vii] Improvement of Main Cognitive Functions in Patients with Alzheimer’s Disease after Treatment with Coconut Oil Enriched Mediterranean Diet: A Pilot Study.  de la Rubia Ortí JE, et al. J Alzheimers Dis. 2018;65(2):577-587. doi: 10.3233/JAD-180184. PMID: 30056419

[viii] Improvement of Main Cognitive Functions in Patients with Alzheimer’s Disease after Treatment with Coconut Oil Enriched Mediterranean Diet: A Pilot Study.  de la Rubia Ortí JE, et al. J Alzheimers Dis. 2018;65(2):577-587. doi: 10.3233/JAD-180184. PMID: 30056419

[ix] Lazarov O, Hollands C. Hippocampal neurogenesis: Learning to remember. Prog Neurobiol. 2016;138-140:1–18. doi:10.1016/j.pneurobio.2015.12.006. PMID: 26855369

[x] Farah BA (2014) Effects of caprylic triglyceride on cognitive performance and cerebral glucose metabolism in mild Alzheimer’s disease: A single-case observation. Front Aging Neurosci 16, 1-4. PMID: 25076901

[xi] Reger MA, Henderson ST, Hale C, Cholerton B, Baker LD, Watson GS, Hyde K, Chapman D, Craft S (2004) Effects of beta-hydroxybutyrate on cognition in memory-impaired adults. Neurobiol Aging 25, 311-314. PMID: 15123336

[xii] How does coconut oil affect cognitive performance in alzheimer patients?  de la Rubia Ortí JE, et al. Nutr Hosp. 2017 Mar 30;34(2):352-356. doi: 10.20960/nh.780. PMID: 28421789

[xiii] Improvement of Main Cognitive Functions in Patients with Alzheimer’s Disease after Treatment with Coconut Oil Enriched Mediterranean Diet: A Pilot Study.  de la Rubia Ortí JE, et al. J Alzheimers Dis. 2018;65(2):577-587. doi: 10.3233/JAD-180184. PMID: 30056419

[xiv] Improvement of Main Cognitive Functions in Patients with Alzheimer’s Disease after Treatment with Coconut Oil Enriched Mediterranean Diet: A Pilot Study.  de la Rubia Ortí JE, et al. J Alzheimers Dis. 2018;65(2):577-587. doi: 10.3233/JAD-180184. PMID: 30056419

[xv] Improvement of Main Cognitive Functions in Patients with Alzheimer’s Disease after Treatment with Coconut Oil Enriched Mediterranean Diet: A Pilot Study.  de la Rubia Ortí JE, et al. J Alzheimers Dis. 2018;65(2):577-587. doi: 10.3233/JAD-180184. PMID: 30056419

[xvi] Improvement of Main Cognitive Functions in Patients with Alzheimer’s Disease after Treatment with Coconut Oil Enriched Mediterranean Diet: A Pilot Study.  de la Rubia Ortí JE, et al. J Alzheimers Dis. 2018;65(2):577-587. doi: 10.3233/JAD-180184. PMID: 30056419

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of GreenMedInfo or its staff.

Candida

Written by Brenton Wight, Health Researcher, LeanMachine
Copyright © Brenton Wight, LeanMachine
Updated 31st January 2020

Candida albicans, also known as Yeast infections, Fungal infections, Mycosis and Thrush

Almost everyone has Candida in their bodies. For most of us, it causes no problems.
But if our immune system is compromised by antibiotics, poor diet, sleep deprivation or other factors, candida can grow uncontrollably.
Each of us has about 70 to 100 or more trillion bacteria in our bodies, weighing between 1 and 2kg, or over 3% of our total body weight.
Most of them good, but some are bad – really bad, like yeast, parasites and other organisms, and most of the time we don’t know they are even there.
If we are healthy with a good diet, our immune system and the good bugs keeps the bad bugs under control. We can never eliminate them, but we can keep them at levels where they can’t do too much harm.
75% of our immune system is in the gut – our trillions of good bacteria is our second, but most important line of defence against unwelcome invaders.
Our first line of defence is in the mouth, and this is where our good bacteria first start to knock out the bad guys, and this is why sugar should be banned from our diet, as sugar keeps the bad bugs fed well.
Healthy gut flora keeps candida under control, because in it’s normal state, Candida is a relatively weak fungal type bug.
Unfortunately, under some conditions, Candida can transform into dangerous mutations, changing to an aggressive bug which can cause systemic (throughout the entire body) destruction.
Candida is very common, and very dangerous.
75% of women, and many men, suffer from Candida in some form at some stage, from minor symptoms including rashes, increased PMS issues and bad breath, to severe conditions like IBS (irritable bowel syndrome), depression, joint pain, and chronic fatigue syndrome.
A Candida problem can be a host of apparently unrelated chronic symptoms which refuse to go away, regardless of treatments, and a course of antibiotics only makes things much worse.

Mainstream Medicine Mistakes

In the 1980’s links were found between candida, chronic fatigue, chronic pain problems and fibromyalgia-type symptoms.
Mainstream health professionals have little idea how to diagnose, let alone treat Candida issues, and those who do attempt to treat Candida use dangerous medications which often exacerbate the situation by destroying our good bacteria, leaving us defenseless against the bad guys.
Creams, oils, lotions, etc are only marginally effective on the skin where applied, and cannot resolve systemic, body-wide infestations.
The “nuke it” approach, with powerful anti-fungal prescription medications, can work temporarily, but Candida is smart enough to disguise itself and eventually develop resistance to the medication.
Drugs used for other purposes such as antibiotics, cortisone therapy, immune-suppressants, even birth control pills can start a Candida infestation explosion.
Candida critters are very smart.
Classified as a fungus, but are in fact part plant, part animal, part bacteria, part parasite, and able to reproduce themselves either sexually (with a mate) or asexually (by themselves), and by re-arranging their own chromosomes, can alter their shape and forms to adapt to the environment and ensure survival.
No surprise that with these attributes, it can be very difficult to dispose of yeast infections!

How Candida Works

In the normal, relatively harmless yeast form, under the microscope, Candida looks like a small white egg, with a smooth and consistent shape and living in our gut.
Candida feeds on the food we eat and normally keeps to itself, but under some conditions, it mutates into a monster super-fungus, multiplying rapidly to colonise in every possible part of the body.
In some areas it can double the colony size every hour, so before long it runs rampant, destroying everything in it’s path.
The best way to start this happening is to feed it it’s favourite food – sugar!

Candida Symptoms

  • White coating on the tongue
  • Development of food sensitivities
  • Aches and pains without a known cause
  • Bloating, constipation or diarrhea
  • Persistent fatigue
  • Dry or other skin problems
  • Vaginitis (inflammation of the female genital area)
  • Breathing problems

Mutated Forms

The mutated form called mycelial-form of Candida grows rhixoids, which are elongated, finger-like tentacles that tunnel, bore and burrow their way through our gut walls, where they enter the blood vessels, to be transported everywhere in the body, blooming to a systemic infection.
They now appear anywhere – heart, eyes, kidneys, liver, lungs and even the brain. A yeast infection is not just a minor annoyance causing a little itching – this is a warning sign that some insidious thing is happening.
Like tree roots which can crack, lift and eventually destroy pavement, Candida does the same thing to our bodies as it mutates into the dangerous super-fungus form. Apart from entering the blood through the cracks in our gut and spreading, another opportunity is now “opened up” – many other undesirable things now enter our blood stream through these cracks; things which should normally stay in our gut: Bacteria, toxins, undigested food, milk, etc.

A short note about Milk

This is a good reason why no-one should ever drink homogenised milk, as the fat globules in homogenised milk are ten times smaller than un-homogenised milk, easily entering the blood through the tiniest holes in the gut.
When undigested milk enters the bloodstream directly through a “leaky gut”, the body’s own immune system recognises this as a unwanted invader and attempts to destroy it. From that time on, the immune system remembers this pattern, and we then have a milk allergy.
Leaky gut is the main culprit for the rapid rise in allergies we have seen in the last few decades, as we are now allergic to every food that passes directly into the blood.
We can find unhomogenised milk at all good supermarkets if we look hard enough, but none of the major brands make it.
Sure we have to shake the container to mix the cream, but that is a small price to pay for our better health, and I should also mention that we should never buy low-fat or skim milk, as all of the nutrients, including CLA (Conjugated Linoleic Acid) and Omega-7, have been lost with the cream.
The “Low Fat” advertising is a gimmick. We lose weight more effectively with full-cream milk compared to low-fat milk.
Milk also contains lactose. Like most other things ending in “ose”, lactose is a sugar, and sugar feeds candida (and cancers).
Again, milk fills a complete chapter in my upcoming book, so I’ll leave a summary: Most people do better without milk, have less allergies without milk, lose more weight without milk, have stronger bones without milk, but if you have to have milk, FULL-CREAM and UN-HOMOGENISED milk is the only reasonably healthy milk to buy, and if you can get it straight from the cow (NON-PASTEURISED), even better.

Candida Waste Products

Candida overgrowth can make us feel sick, tired, plain lousy, and can make us drunk from inside out!
As yeast dies as part of it’s natural life cycle, acetaldehyde is released. This is a toxin which the liver converts to ethanol, the same hangover-causing chemical produced by high consumption of alcohol, so Candida sufferers often feel like they have a hangover without drinking a drop of alcohol, because they have an alcohol factory in their own body.
Another problem, as if we don’t have enough problems already, is “Candida Poo”, which is the 79 distinct toxins and waste by-products released as part of the Candida lifecycle. It’s bad enough that we feed all parasites, but we have to clean up and dispose of their waste and toxic elements as well as recover from the damage and disease they cause!
Candida infestation can continue growing, producing more acetaldehyde, causing our blood alcohol level to rise enough to cause symptoms such as impaired thinking, lack of concentration, irritability, depression, brain fog, slowed reflexes, fatigue and other signs of excess alcohol.
In this state, red blood cells have trouble flowing into small capillaries, aggravating migraine headaches, fatigue, muscle aches, slowing healing.
At the same time, white blood cells have reduced ability to fight infection, often causing allergies, rashes, acne and even further inhibiting of healing processes.
Like the way bread rises from yeast because of carbon dioxide released from carbohydrate fermentation, Candida builds up carbon dioxide in the intestine, resulting in excess gas, bloating, and yet another waste product for the over-taxed body to dispose of.

The Real Candida Cause

Can we defeat this little terror? Of course we can, but we must get back to basics to eliminate problems once and for all.
Why do we get Candida in the first place? Everyone has Candida, because we can’t help consuming it from food, water and the environment, but the reason it develops is because our body gets out of balance, and our immune system gets compromised, allowing immediate, almost uncontrollable flourishing of Candida.
We cannot destroy Candida completely without killing ourselves, so our only real, long-lasting solution is to re-balance the body, restore the immune system, and let the body’s own natural defenses do their job.
If we have bad breath, rashes, fatigue, trouble concentrating, brain fog, strange allergies, weird pains or anything we can’t explain, it could be a sign that something is out of whack, allowing Candida to get out of control, and even if the problem is not Candida, correcting the balance and immune system of the body is the first step to recovery from any sickness.

Candida and Cancer

New research is now finding a link between Candida infections and Cancer. While billions of dollars are being spent on cancer research, cancer cases are increasing, now overtaking cardiovascular issues as a leading cause of death. LeanMachine suggests that staying Candida free is the first step in staying Cancer free. Like most modern diseases, prevention is the key, then requiring no cure.

The Cure

We must create an environment which is good for nourishment of the body, but bad for Candida. By following these guidelines, we will remain Candida free, and also reduce the risk of Cancer, Diabetes, Alzheimer’s, and most other “modern” diseases.

  • Eliminate all sugar from the diet. The natural sweetener Xylitol is OK as it is a 5-carbon sugar alcohol, and despite it’s misleading name (it is technically neither a sugar, nor an alcohol), it will not feed Candida, and has far less insulin-spiking effect. Other alternatives are Erythritol and Stevia, with virtually zero insulin spiking. Avoid all artificial sweeteners. In the long term, lose the sweet-tooth altogether
  • Change to an alkaline-forming diet, as acids cause Candida and other parasites to thrive. See my Alkaline Diet Article
  • Avoid yeast in all forms. All grain/flour based products will acidify and aggravate our bodies, and are usually made with yeast, the best food for Candida. No more bread, cakes, biscuits, muffins, etc. Tough for some people but you will thank me a few weeks! Excess weight will slowly disappear, while digestion, health and energy will all improve, and the holes in the gut caused by flour products will begin to heal
  • Keep cool and dry, wear well-ventilated clothing and live in a well-ventilated house. Candida loves sugar, warmth and moisture. Avoid artificial fibres in clothing. Keep to natural fibres like cotton, bamboo or wool. Before buying any clothing, give it a sniff. Any trace of a chemical smell? Leave it in the store
  • Never take antibiotics unless there is a severe, life-threatening situation. Antibiotics encourage Candida and other harmful organisms by destroying their natural enemies (the good bacteria)
  • Reduce carbohydrate intake, as all carbs feed Candida. This means potatoes, pasta, grains, cereals, breads, cakes, etc. A healthy body requires NONE of these “foods”. There is NO medical condition such as “Carbohydrate Deficiency” as the body can make all the carbs it needs from breaking down protein
  • Take liver detox supplements such as Milk Thistle
  • Add some parsley to each meal. Easy to grow yourself in a pot, and available at the greengrocer or supermarket
  • Take a supplement containing Pau D’Arco or Candida Support
  • The main ingredient in Pau d’Arco is beta-lapachone, also a potent catalyst for NADH (Reduced Nicotinamide Adenine Dinucleotide, or NAD+ or CoEnzyme E1), important for mitochondria, the energy components of every cell in the body and as a sensor for stress and disease
  • Also consider Pumpkin Seed Oil for more parasite protection
  • Most kitchens have cloves in the spice rack. Add them to everything to help control Candida and other parasites, and add a great flavour at the same time
  • Berberine has anti-inflammatory, immune-enhancing, anti-microbial properties against bacteria, protozoa, and fungi. Berberine’s action against Candida pathogens is stronger than prescription antibiotics commonly used
  • Echinacea is also of benefit in building the immune system
  • Aloe contains a mucopolysaccharide with strong immune properties and also benefits the mitochondria
  • Take a probiotic supplement such as Acidophilus or at least eat some yogurt every day to help re-build the good gut flora
  • Try to use a probiotic that includes a FOS (fructo-oligosaccharides), a pre-biotic that enhances functionality of gut flora, included in Acidophilus above
  • Fermented foods – sauerkraut, etc are a good source of probiotics, generally much more than available in supplements
  • Eat more high-fibre foods like chia seeds, preferably containing over 40 grams of fibre daily
  • Or take a daily fibre supplement containing Psyllium to improve regularity, keep things moving through the colon, improve internal cleansing and reduce risk of colon cancer. Can help relieve constipation, but because it increases bulk with both soluble and insoluble fibre, may also help with diarrhoea
  • Eat some garlic every day, or at least onions or shallots, but if the taste and odour is not to your liking, then try Odorless Garlic
  • To help cleanse the blood and the liver, eat Chlorophyll-rich foods (Green leafy vegetables) every day, or take Chlorophyll supplements. The molecular structure of Chlorophyll and Haemoglobin appear identical, apart from one small difference: Haemoglobin has a central iron atom (making blood red), while Chlorophyll has a central magnesium atom (making plants green)
  • Coconut Oil contains Caprylic Acid, along with capric acid and lauric acid, MCT (Medium Chain Triglycerides) that help defeat candida as it is a natural anti-fungal, anti-viral and anti-bacterial product, and also helps lose weight, as MCT’s go straight to the liver to be burned as fuel and cannot be stored as fat
  • Avoid antibiotics, steroid hormones and oral contraceptives, as they alter the gut bacteria, increasing risk of candida changing into an invasive form
  • Avoid antibiotics in food such as from animals raised in concentrated animal feeding operations which are fed antibiotics to counteract bad sanitation and to boost growth. Ask for “grass fed” or “pasture raised” meat, and limit meat consumption (acidic) in favour of plant foods (alkaline)
  • Get more good sleep, more exercise and an alkaline, sugar-free diet, but never consume food or drink with aspartame or other artificial sweeteners, which destroy good gut bacteria. Stevia is a better natural choice and has anti-cancer properties
  • Treat the partners of infected patients, even if there are no symptoms, as they can also be carrying abnormally large amounts of Candida, potentially leading to constant cross-infections
  • Continue treatment until all symptoms have disappeared, plus a further 2 weeks, which destroys eggs laid by the previous generation

Drug Therapy

There are some prescription drugs which can help if all of the above fails and the condition is serious:

  • Nystatin, an antifungal, available as tablets or a powder, and fairly benign with few side effects.
  • Diflucan, an antibiotic if all else fails

LeanMachine Online Supplements: Health Supplements

Updated: 31st January 2020.   Copyright © 1999-2020 Brenton Wight – Lean Machine

All About Ginkgo Biloba: Benefits of This Timeless Herbal Supplement


Reproduced from original article:
https://articles.mercola.com/vitamins-supplements/ginkgo-biloba.aspx

Ginkgo biloba leaves

STORY AT-A-GLANCE

  • Known as one of the oldest trees on the planet, ginkgo biloba has been long revered for its many medicinal uses
  • Get to know more about ginkgo biloba – how it works, what’s the best way to take it and other health considerations before using this supplement

The ginkgo biloba tree is known for its resilience and hardiness, and for good reason – it is the oldest living tree species known to man. It’s said that a ginkgo tree can live for as long as 1,000 years, with heights reaching up to 120 feet.1 Thus, it should come as no surprise that this plant can have healing benefits, too.

Today, ginkgo biloba ranks as one of the top-selling herbal remedies,2 with a long history of medicinal use for various illnesses and health problems. Get to know more about ginkgo biloba – how it works, what’s the best way to take it and other health considerations before using this supplement.

What Is Ginkgo Biloba?

Also known as the maidenhair tree, ginkgo biloba is one of the oldest trees on Earth, having said to have been in existence for 350 million years.3 In fact, it’s often referred to as a “living fossil,” as it has continued to thrive even after major extinction events.4

The ginkgo biloba tree is the only surviving species from the Ginkgoaceae family.5 The name is said to come from the Japanese words “gin” and “kyo,” which means “silver” and “apricot” respectively, which refers to the ginkgo fruit’s resemblance to apricots.6

Ginkgo biloba is native to Asia, particularly in China, Korea and Japan. In fact, some ginkgo trees in China are said to be 2,500 years old.7 This plant is also believed to have thrived in Europe and North America during ancient times, but after the Ice Age, all the wild ginkgo trees in these areas were destroyed.8

The ginkgo tree is known for being tough and hardy – it can even thrive in polluted environments, such as urban roadsides and large modern cities.9 The plant has short branches and fan-shaped leaves, which change colors during fall.

However, ginkgo fruits are inedible, emit an unpleasant scent (reminiscent of rancid butter10) and have inner seeds that may be poisonous.11 Today, organic ginkgo biloba supplements are widely available, and is sold either in liquid extract, capsule or tablet form. The extract is made from the dried leaves of the plant.12

Ginkgo Biloba’s Uses for the Body: Its Mechanism of Action

Although it has been popularly utilized in Chinese medicine for more than 5,000 years, it is only recently that researchers are uncovering what components account for ginkgo biloba’s many uses and benefits. According to the book “Prescription for Herbal Healing,” written by Phyllis A. Bach:13

“Ginkgo increases the body’s production of adenosine triphosphate (ATP), a compound that is the main source of energy at the cellular level. This activity has been shown to boost the brain’s metabolism of glucose for energy and to increase its electrical activity.”

Particularly impressive are the flavonoids in ginkgo, called ginkgolides, which are said to have an effect on even the smallest microcapillaries, leading to widespread benefits on all of the body’s organs, especially the brain.14 Ginkgo biloba is also known for its antioxidant abilities, brought on by ginkgolides as well, and is said to prevent platelet aggregation inside arterial walls to keep plaque from forming.15

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Take a Look at Some of Ginkgo Biloba’s Health Benefits

So what exactly is ginkgo biloba good for? According to research, taking ginkgo biloba supplement may help improve:

Memory and thinking — Some research suggests that ginkgo may offer improvements in memory, speed of thinking and attention among healthy adults.16 In fact, it is touted as a “brain herb” that may have potential benefits for people suffering from dementia.17

Eye health — Ginkgo may help prevent macular degeneration and glaucoma by deactivating the platelet-activating factor (PAF), which is necessary for growth of new capillaries, and increasing circulation within the eye, supplying more oxygen to the retina.18 One small study found that people with glaucoma had improvements to their vision after taking 120 milligrams of ginkgo daily for eight weeks.19

Reproductive health and sexual function — One of ginkgo biloba’s benefits for men is its potential for helping treat impotence. It enhances nitric oxide’s effects, allowing more blood to flow into the penis to help maintain erection during sexual intercourse.20

Anxiety — According to a study published in the Journal of Psychiatric Research, people with generalized anxiety disorder experienced better anxiety relief after taking ginkgo biloba, compared to those who took a placebo.21

Raynaud’s syndrome — This is an illness that triggers episodes of numbness and cold due to a decrease in the blood supply to the fingers and toes (earlobes, lips and nose may also be affected), triggered by stress or cold temperatures.22 A study said that ginkgo biloba may reduce the number of episodes in patients who have this illness.23

Skin and hair health — The rich antioxidant content of ginkgo biloba can help eliminate free radicals that can cause wrinkles and other signs of aging. Extracts of this herb are also used in hair care products to keep your tresses strong, long and shiny.24

Ginkgo Biloba Studies Claim It May Benefit Dementia and Alzheimer’s Patients

There have been numerous research conducted on the effectiveness of ginkgo biloba for certain health conditions, and one of the most widely studied claims is its potential effects for degenerative illnesses like dementia and Alzheimer’s disease. According to WebMD, this herb may halt the progression of dementia symptoms, especially if the condition is brought on by atherosclerotic vascular disease.25

Initially, ginkgo was thought to improve blood flow going to the brain, but recent findings reveal that this herb may actually give protective effects to nerve cells against Alzheimer’s as well.26 One study, for example, found that EGb 761, a ginkgo biloba extract, may be clinically effective in treating Alzheimer’s dementia.27

Another research supported this claim, saying that the extract was safe to use and may help stabilize and improve cognitive and social functioning of dementia patients for between 6 to 12 months.28 However, there’s still conflicting evidence on whether it can have the same cognitive effects on healthy adults.

Ginkgo Biloba May Have Side Effects

There are individuals who reported having mild upset stomach and mild headaches that last a day or two after taking ginkgo biloba. Some who took large doses experienced nausea, vomiting, diarrhea, restlessness and dizziness. Finnish physicians also reported that their patients experienced orthostatic hypotension, a sudden loss of blood pressure when standing up, after using ginkgo for several days.29

In addition, the leaves of this herb contain long-chain alkylphenols, which have allergenic properties. If you are allergic to poison ivy and other plants with alkylphenols, do not take ginkgo.30

Take Note: Listen to Your Body When Using Ginkgo Biloba

As with any supplement, it is important that you listen to your body when using ginkgo biloba. It may offer potential benefits, but if your body is exhibiting unpleasant symptoms while using this supplement, then you may be better avoiding it or finding another alternative. Seek your physician’s advice should any of the symptoms above occur.

Why Hard Water Decreases Heart Attacks


Reproduced from original article:
https://articles.mercola.com/sites/articles/archive/2020/01/20/magnesium-linked-to-better-heart-health.aspx

Analysis by Dr. Joseph Mercola     Fact Checked image

STORY AT-A-GLANCE

  • Magnesium insufficiency has been linked to a higher risk for high blood pressure, cardiovascular disease, arrhythmias, stroke and sudden cardiac death
  • Research has shown people drinking soft water on a regular basis are more susceptible to lethal arrhythmias (irregular heartbeat) than those drinking hard water. One hypothesis that can account for this is magnesium deficiency, as hard water contains higher levels of magnesium than soft water
  • Magnesium supports heart health by combating inflammation (which helps prevent high blood pressure and hardening of your arteries) and improving blood flow by relaxing your arteries and preventing your blood from thickening
  • Magnesium also plays a role in the creation of adenosine triphosphate (ATP), the energy currency of your body. Being a heavy energy user, your heart needs sufficient amounts of ATP to function properly
  • Magnesium also affects your mitochondrial function and health, as it’s required both for increasing the number of mitochondria in your cells and increasing mitochondrial efficiency

When it comes to heart health, one of the most influential nutrients is magnesium. While required for the healthy function of most cells in your body, magnesium is particularly important for your heart, kidneys and muscles.

As far back as 1937, researchers warned that low magnesium levels pose serious risks to the heart, and that it may actually be the most significant predictor of heart disease.1 More recent research suggests even subclinical magnesium deficiency can compromise your cardiovascular health.2

Importantly, your mitochondria require magnesium to produce ATP. It’s also required for the metabolic function of your cells and the activation of vitamin D.3,4 All of these are important for healthy heart function.

It also supports heart health by relaxing your blood vessels, normalizing blood pressure, lowering inflammation and supporting endothelial function (the cells’ lining the interior of your blood vessels).5

Hard Water Linked to Better Heart Health

To celebrate its 175th anniversary, Scientific American recently took a look back into its archives, publishing a short summary of research presented in its June 1969 issue:6

“Several studies in the past decade have suggested that the death rate from coronary disease is inversely correlated with the hardness of the local water supply: the harder the water, the lower the coronary rate.

A study7 recently published in the New England Journal of Medicine reports evidence that the excess coronary deaths in soft-water areas are almost entirely sudden deaths …”

By reviewing the death certificates of 55,000 individuals who died from heart-related issues in Ontario during 1967, and then correlating the deaths according to the hardness of the local water supply, the Canadian researchers were able to conclude that people drinking soft water on a regular basis were more susceptible to lethal arrhythmias (irregular heartbeat). What might explain this curious correlation?

One theory has focused on the magnesium level found in the water.8 Soft water is lower in magnesium than hard water, thus making you more prone to magnesium deficiency. (The very definition of hard water is that it contains a high concentration of dissolved metals — calcium and magnesium in particular.9,10)

According to a 2002 study,11 magnesium-rich mineral water can contribute between 6% and 17% of your total daily magnesium intake. That said, a 2013 paper12 in the International Journal of Preventive Medicine points out that the exact mechanisms responsible for the relationship often found between harder water and lower cardiovascular risk has yet to be ascertained.

The higher magnesium level in hard water appears to be a promising hypothesis, though, and several studies point to magnesium-rich water being an important factor. As noted in the International Journal of Preventive Medicine, which cites a number of such studies (as well as some in which this relationship was not found):13

In a Swedish study, the skeletal muscle magnesium levels were a significantly higher in persons living in an area with a higher water magnesium.”

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Even Subclinical Magnesium Deficiency Can Be Problematic

As mentioned, magnesium supports heart health through a number of different mechanisms.14 For starters, it combats inflammation, which helps prevent high blood pressure and hardening of your arteries. It also improves blood flow by relaxing your arteries and preventing your blood from thickening.

Magnesium also plays a role in the creation of adenosine triphosphate (ATP), the energy currency of your body.15,16 Needless to say, without sufficient energy, cellular functions throughout your body will suffer, creating a cascade of dysfunction. Your heart in particular, being a very heavy energy user, needs sufficient amounts of ATP to function properly.

Magnesium also affects your mitochondrial function and health, as it’s required both for increasing the number of mitochondria in your cells and for increasing mitochondrial efficiency.

Basic effects such as these can account for why magnesium insufficiency has been linked to a higher risk for high blood pressure,17 cardiovascular disease, arrhythmias, stroke18 and sudden cardiac death.19

A 2018 paper20 in the Open Heart journal also warns that even subclinical deficiency can result in heart problems, and that most people need at least 300 milligrams more magnesium per day than the current recommended dietary allowance prescribes. According to the authors:

“… While the recommended … dietary allowance for magnesium (between 300 and 420 mg /day for most people) may prevent frank magnesium deficiency, it is unlikely to provide optimal health and longevity, which should be the ultimate goal.”

The theory that we may need more magnesium than is currently recognized is also supported by a 2016 meta-analysis,21 in which all-cause mortality was lowered by 10% simply by increasing magnesium intake by 100 mg per day.

Magnesium Is Important for Brain Health Too

Magnesium is also important for brain health and the prevention of dementia. Memory impairment occurs when the connections (synapses) between brain cells diminish. While many factors can come into play, magnesium is an important one. As noted by Dr. David Perlmutter, a neurologist and fellow of the American College of Nutrition:22

“It has now been discovered that magnesium is a critical player in the activation of nerve channels that are involved in synaptic plasticity. That means that magnesium is critical for the physiological events that are fundamental to the processes of learning and memory.

As it turns out, one form of magnesium, magnesium threonate, has the unique ability to permeate the brain and enhance the receptors that are involved in this process.”

The specific brain benefits of magnesium threonate were demonstrated in a 2010 study23 published in the journal Neuron, which found this form of magnesium enhanced “learning abilities, working memory, and short- and long-term memory in rats.” According to the authors:24

Our findings suggest that an increase in brain magnesium enhances both short-term synaptic facilitation and long-term potentiation and improves learning and memory functions.”

Magnesium is also a well-recognized stress reliever,25 and by catalyzing mood-regulating neurotransmitters like serotonin, it helps prevent anxiety and depression.26

Research27 published in 2015 found a significant association between very low magnesium intake and depression, especially in younger adults. A study28 published in PLOS ONE demonstrated magnesium supplementation improved mild-to-moderate depression in adults, with beneficial effects occurring within two weeks of treatment.

How to Assess Your Magnesium Status

When it comes to measuring your magnesium level, your best bet is an RBC magnesium test, which measures the amount of magnesium in your red blood cells. Tracking any symptoms of magnesium deficiency is also recommended, as your need may be higher or lower depending on your lifestyle and health status.

Common signs and symptoms of magnesium insufficiency include but are not limited to the following.29,30 A more exhaustive symptom’s list can be found in Dr. Carolyn Dean’s blog post, “Gauging Magnesium Deficiency Symptoms.”31

  • Seizures, muscle spasms (especially “charley horses” or spasms in your calf muscle that happen when you stretch your leg), eye twitches and or numbness or tingling in your extremities
  • Insulin resistance
  • High blood pressure, heart arrhythmias and/or coronary spasms
  • Increased number of headaches and/or migraines
  • Low energy, fatigue and/or loss of appetite

The “Trousseau sign”32 can also be used to assess your magnesium status. To check for this sign, a blood pressure cuff is inflated around your arm. The pressure should be greater than your systolic blood pressure and maintained for three minutes.

By occluding the brachial artery in your arm, spasms in your hand and forearm muscles are induced. If you are magnesium deficient, the lack of blood flow will cause your wrist and metacarpophalangeal joint to flex and your fingers to adduct. For a picture of this hand/wrist position, see Figure 1 in the paper “Trousseau Sign in Hypocalcemia.”33

Would You Benefit From Magnesium Supplementation?

A number of studies suggest magnesium insufficiency or deficiency are extremely common, both among adults34 and teens,35 in part due to the fact that most people eat a plant-deficient diet. Magnesium is actually part of the chlorophyll molecule responsible for the plant’s green color.

However, even if you eat plenty of greens, you may still not get enough, thanks to most soils being so depleted of minerals. Your body’s ability to absorb magnesium is also dependent on having sufficient amounts of selenium, parathyroid hormone and vitamins B6 and D.

Absorption is further hindered by excess ethanol, salt, coffee and phosphoric acid in soda, and things like sweating, stress, lack of sleep, excessive menstruation, certain drugs (especially diuretics and proton-pump inhibitors), insulin resistance and intense exercise can deplete your body of magnesium.36,37

Research shows just six to 12 weeks of strenuous physical activity can result in magnesium deficiency,38 likely due to increased magnesium demand in your skeletal muscle.

For all of these reasons, most people probably need to take supplemental magnesium. The RDA for magnesium is around 310 to 420 mg per day depending on your age and sex,39 but many experts believe you may need a minimum of 600 mg per day.40

I suspect many may benefit from amounts as high as 1 to 2 grams (1,000 to 2,000 mg) of elemental magnesium per day, as the extra magnesium may also help mitigate unavoidable exposures to electromagnetic fields (thanks to its calcium channel blocking effect). To learn more about this, see my previous article on how to reduce EMF exposure.

You can easily improve your magnesium status with an oral magnesium supplement. My personal preference is magnesium threonate, as it appears to be the most efficient at penetrating cell membranes, including your mitochondria and blood-brain barrier. You can learn more about this in “Cognitive Benefits of Magnesium L-Threonate.”

Magnesium Testing Is a Valuable Health Screen

Considering the importance of magnesium for good health — including cognition and heart health — it’s a good idea to measure your level. GrassrootsHealth Nutrient Research Institute, which has spearheaded research into vitamin D and omega-3, now also offers low-cost testing for magnesium.

Join Magnesium* Plus Focus Project today!

Like its vitamin D and omega-3 projects, the Magnesium*PLUS Focus Project41 aims to identify the ideal dosage and level, the specific health outcomes associated with magnesium deficiency and sufficiency, the dose-response relationships and much more. As noted by GrassrootsHealth:42

“Measuring your nutrient status, adjusting intake as needed, and re-testing is the only way to tell if your nutrient intake is helping you achieve sufficient or desired nutrient status which is tied to particular health outcomes.

We will analyze the collected data and give participants feedback on how the magnesium could be working for them; we will publish scientific papers on key results, the first after meeting an enrollment target of 1,000 participants. There will be preliminary analyses and interim newsletters available for all during the enrollment phase.”

Adding the “Plus Elements” test to this magnesium test will also measure your selenium, zinc and copper levels, important trace elements that interact with magnesium, as well as three toxic heavy metals (lead, cadmium and mercury) that can interfere with and block availability of these essential elements.

Magnesium* Plus Elements Project

Alzheimer’s Prevention

Written by Brenton Wight, researcher and LeanMachine

Copyright © Brenton Wight, LeanMachine

Doctors say there is no cure for Alzheimer’s Disease, in spite of over 80 billion dollars in research over the last few decades.
This is partly true, as there is no drug, no “magic bullet” to slow or stop this dreadful condition.
Hundreds of studies with new drugs have shown most of the time that those on a placebo did BETTER than those on the drug!
In rare cases, those on the drug did very slightly better, but any improvement was not enough to justify bringing the drug to market.
However, we CAN identify risk factors, and we CAN in most cases prevent the onset of Alzheimer’s, and we CAN in most cases reverse the disease, or at least ease the symptoms to give the patient and the carers a better quality of life.
If the intervention is soon enough, it CAN be CURED in some, but not all cases.
There is no miracle one-shot treatment, but a combination of many factors.
The time to start treatment is not when we are 60 and forget where the keys are, but from birth!
The lifetime changes we need to prevent Alzheimer’s will also prevent heart disease, diabetes, cancer and many other diseases, and give our lives vitality.

How many people are at risk?

In the USA, over 5 million Americans have Alzheimer’s disease, and around 14% of the population will eventually get Alzheimer’s, or around 45 million people.
Results in Australia are similar. Over 10% of the population over 65 have Alzheimer’s, and 30% of those over 85 have Alzheimer’s. In the decade from 2010 to 2020, deaths from Alzheimer’s has risen 20% and looks set to replace Cardiovascular disease as the Number 1 cause of death.
Many people now suffer from Early Onset Alzheimer’s, showing signs as young as 30 years of age.
In the USA, it is now the third leading cause of death, but these figures are understated. People do not actually die from Alzheimer’s – they die because the parts of the brain that control bodily functions shut down, so they die when their organs shut down.
The patient may die from pneumonia because the lungs now cannot function or some other organ fails to work and the Doctor or Coroner has to determine which organ failed.
This is a problem in every country, but some countries have very much reduced rates of Alzheimer’s, mainly due to better diets and reduced toxins.

Diagnosis

Originally, there was no firm diagnosis without examining the brains of patients after death.
Researchers found that most patients had Amyloid Plaques in the brain, and also high levels of aluminium.
PET scans (Positron Emission Tomography) are used with a radioactive tracer (which binds to amyloid plaques) to determine the amount and location of amyloid plaques in the brain.
However, this diagnosis is still not conclusive, as many people have amyloid plaques, but no sign of any dementia even into old age, although these people have a higher risk. Often symptoms do not appear for decades after the start of amyloid plaque deposits. Other patients have no sign of Amyloid plaques but still have Alzheimer’s, so drugs developed to reduce Amyloid plaques have proven unsuccessful in prevention and treatment.
Standard blood tests for glucose level, triglycerides, kidney and liver function can help determine the risk. However, those with less than optimum blood results may die of Cardiovascular, Cancer or some other disease before Alzheimer’s sets in.
So the PET scan is used with other tests for cognitive performance to arrive at a diagnosis.

Who is at risk?

Genetics plays an important part, and so does diet, exercise, lifestyle and supplements.
Here are some risk factors, in no particular order:

  • Age is the greatest risk factor. Dementia can affect about 10% of those over the age of 65, but 33% of those over 80
  • Gender – Women represent over 60% of Alzheimer’s patients, but part of this may be due to their longer lifespans
  • Gluten – Celiacs often have “Wheat Brain” causing disturbances, anxiety, depression and Alzheimer’s. Many dementia patients recover fully on a gluten free diet
  • Prescription medications such as many sedatives, hypnotics, blood pressure, hay fever, insomnia, depression and arthritis medications are linked to higher risk of Alzheimer’s
  • Anaesthetics are linked to Alzheimer’s. The more operations people have, the higher the risk
  • High Blood Pressure (systolic over 140 in mid-life) doubles the risk of Alzheimer’s and increases vascular dementia by 600%, but blood pressure medications can be just as bad, so reduce it naturally without medication
  • Sleep Apnea starves the brain of vital oxygen and increases risk of Alzheimer’s
  • B-12 deficiency increases Alzheimer’s risk. Gastric Bypass Surgery, Celiac disease, vegan/vegetarian diets, antacids (like Nexium) and many medications all reduce availability and/or absorption of B-12
  • Diabetes doubles the risk of Alzheimer’s (often called “Diabetes of the Brain” or “Type 3 Diabetes”)
  • Vision problems increase Alzheimer’s risk. Opthalmologists can detect abnormal widths of blood vessels in the retina which can indicate early Alzheimer’s
  • Tobacco – Smokers have double the risk for Alzheimer’s. Family and others breathing second-hand smoke also have higher risk
  • Living alone after a partner’s death means we have six times the risk of Alzheimer’s, and those who divorce and live alone have three times the risk.
  • Isolation is a significant risk factor for depression and dementia. Find a friend!
  • Obesity is a risk. The lower the BMI (Body Mass Index) the lower the risk. Obesity raises risk by around 75%
  • Family history increases the risk. See the Genetics section below, but environmental factors, diet and lifestyle choices can be passed on to children
  • Education improves outcome, and lack of education increases Alzheimer’s risk. Studies suggest higher education increases “cognitive reserve” which may offset dementia symptoms
  • Concussion or head trauma increases Alzheimer’s risk exponentially with the number and severity of head injuries
  • Quality sleep is essential for the ability of the body to repair itself by flushing toxins from the brain
  • Excessive alcohol consumption can lead to alcoholic dementia and higher risk of Alzheimer’s as well as many other health risks
  • Mental activities improves the brain, physically and psychologically. Learn new things strengthens and develops new nerve cells
  • Sedentary lifestyles are a large risk for the brain as well as the body. Exercise is a must for the brain and the body
  • Chronic bladder disease increases risk
  • Chronic Candida infections increase risk

Overcoming risk factors:

  • Change the diet – see below
  • Get regular, uninterrupted sleep
  • Socialising, visiting friends, joining a group
  • Crosswords, puzzles, new experiences, learning a musical instrument or another language
  • Exercise helps control blood glucose levels, keeps excess weight down, increases oxygen and circulation, and joining a gym can also help with socialisation
  • Use the many supplements available

Genetics

There is a strong genetic predisposition to Alzheimer’s, but also there is a strong contribution of environment, diet and lifestyle.
Rates of Alzheimer’s disease have increased much faster than any genetic changes could have occurred.
This means that much is under our control, because even with a genetic predisposition, we can reduce risk with epigenetic (non-genetic influences on gene expression) changes.
Example: The most important genetic risk factor is the ApoE epsilon 4 allele (ApoE4), and 14% to 18% of the population has this gene.
Everyone carries two copies of the APOE gene, which makes the protein ApoE (apolipoprotein E).
There are three different types (alleles) of the APOE gene: E2, E3 and E4, and because we all have two copies of the gene, the combination determines our APOE “genotype” which can be any combination of the 2 copies: E2/E2, E2/E3, E2/E4, E3/E3, E3/E4, or E4/E4.
The majority of people have two E3 alleles (E3/E3) so this is defined as the “average risk”.
The E2 allele is the least common form, and if we have two E2 alleles (E2/E2) or one E2 and one E3 (E2/E3) we have about 40% REDUCED risk of Alzheimer’s.
The E4 allele, present in 14% to 20% of the population, increases the risk for Alzheimer’s, especially late-onset Alzheimer’s, but this does NOT mean that we will get Alzheimer’s disease if we have one or two copies of E4, as about one third of Alzheimer’s patients do not have even a single E4.
All it means is that our risk is increased, also increased is the risk of potential Alzheimer’s at a younger age.
To quantify the risk:
If we have no copies of E4, we still have around 9% risk of Alzheimer’s.
If we have a single copy of E4, our risk increases to around 30%.
If we have two copies of E4, risk is between 50% to 90% but in all cases, we CAN REDUCE the risk.
Many people are horrified to learn that they have up to a 90% risk of Alzheimer’s, but they need not be.
With some dietary, lifestyle and supplement changes, those at greatest risk can easily fall into the 10% who do NOT get Alzheimer’s.

SAD (Standard American Diet)

Genetic statistics above apply only to average people, typically Caucasians living in the Western World and consuming a typical Western diet of processed food, sugar, MSG, hydrogenated oils, chemicals, heavy metals, pesticides, insecticides and other toxic substances.
These statistics do NOT apply to those with a healthy diet of natural, organic food living in a low-toxin environment.
In fact, many people already down the cognitive decline have recovered on a healthy diet and sustained the improvement for several years, according to Dr Dale Bredesen who has been running a program for years now.
Dr Bredesen does not know how many more years it will be, but does know that patients on the program have removed the biochemical drivers which can be measured in blood tests, so so is very optimistic about their future health for many years to come.

Should we get genetic testing?

This is up to the individual. Some people would prefer not to know. Others want to know.
My father died from Alzheimer’s at about age 72 after many years in a Nursing Home, existing but without knowing who his family members were. So did my Grandmother on my Mother’s side, so I assume I may well have inherited a high genetic risk. I am now 73 as I revise this article. For me, testing is irrelevant, because I changed to a Paleo-style diet at age 63, which turned my life around.
From obese to lean, from grey hair to brown, from allergies to everything to allergies to nothing, from high blood pressure and triglycerides to normal, from poor physical strength to strong, fit and full of energy, from frequent headaches to none, from always getting sick to never getting sick.
If I had the genetic test and it was the worst result, I would only continue to do what I am doing now, using dietary and lifestyle modifications.
Have I halted Alzheimers? I hope so, but I often cannot remember some of the thousands of medical terms I have come across in my 10 years of research.  Come back here in 27 years as I approach 100 and I will let you know how I have done.

Amyloid Plaques vs Tangles

Amyloid is a protein, normally found throughout the body. In Alzheimer’s, this protein divides improperly, creating beta amyloid which is toxic to brain neurons.
Amyloid is actually antimicrobial and has benefits for the body, but some people, especially those with the E4/E4 alleles cannot naturally break down these plaques, but there are dietary methods which can.
Not all Alzheimer’s patients have beta Amyloid plaques. About 10% of patients have neurofibrillary tangles which cause similar symptoms, but are also inclined to have more aggressive behavior.

Three Kinds of Alzheimer’s

Humans liberate amyloid as a protective response in the body to three different fundamental metabolic and toxic perturbations:

  • Type 1: Characterized by systemic inflammation. Blood tests typically reveal high hs-CRP (high-sensitivity C-reactive protein), low albumin:globulin ratio,
    and high cytokine levels such as interleukin-1 and interleukin-6. Imaging reveals temporoparietal reductions in glucose utilization.
    Those at risk include people with chronic infections or inflammation from other causes, and the normal antimicrobial protective response liberates amyloids
  • Type 2: Characterized by normal inflammation, but an atrophic (wasting away) profile, with reduced support from estradiol, progesterone, testosterone, insulin, and vitamin D, often with high homocysteine and insulin resistance. Imaging reveals temporoparietal reductions in glucose utilization. As NGF (Nerve Growth Factor) diminishes, amyloid production increases.
    Type 2 in particular can be CAUSED by LOW cholesterol, resulting in atrophy (brain shrinkage), reduced hormone production, poor health and eventually Alzheimer’s.
    All because we are taking statins that lower cholesterol, or we are not eating enough healthy fats.
    We prevent our cells from doing what they are supposed to do, so we end up with a shrunken brain without the lipid (fat) content we need. A fat-free diet means atrophy of the brain.
    See the Cholesterol Fraud and the Big Fat Lie sections below.
  • Type 3: Different from types 1 and 2. Still β-amyloid positive and phospho-tau positive), but a younger onset (late 40s to early 60s).
    Genotype ApoE is usually E3/E3 instead of E4/E4 or E3/E4 with little or no family history.
    Onset usually follows a period of stress, depression, sleep loss, anesthesia, or menopause/andropause.
    Memory loss is not a main symptom, instead there are cortical issues: dyscalculia (trouble with arithmetic), aphasia (trouble speaking or understanding speech – damage to the left side of the brain),
    executive dysfunction (emotional or behavioural problems from frontal lobe issues).
    Imaging studies often reveal extra-hippocampal disease, greater general cerebral atrophy and frontal-temporal-parietal abnormalities.
    Lab results often reveal hypozincemia (low zinc) and/or a high copper:zinc ratio, and can indictate adrenal fatigue
    (low pregnenolone, DHEA-S (dehydroepiandrosterone sulfate), and/or AM cortisol. Chronic infections like mycotoxins, Lyme, viral infections, HSV-1 (a herpes simplex virus) are all risk factors


Some patients have “Alzheimer’s type 1.5” where a combination of symptoms of both type 1 and 2 Alzheimer’s occurs.
Glycotoxicity (too much sugar in the brain) causes an insulin resistant brain. Combine this with AGEs (Advanced Glycation End products), and we have both inflammation from AGEs, plus atrophic withdrawal response because we are now resistant to insulin.
So we have a double condition of type 1 and type 2.

Type 3 patients often have MARCoNS (Multiple Antibiotic-Resistant Coagulase-Negative Staph), a colonisation of antibiotic-resistant staphylococcus in the nasal cavity.
Also high blood levels of TGF-beta-1 (Transforming Growth Factor beta-1), high C4A (a protein that in humans is encoded by the C4A gene), and low MSH (Melanocyte-Stimulating Hormone) is very common, typically with HLA-DR/DQ haplotypes shown by Dr Ritchie Shoemaker to be associated with CIRS.

Alzheimer’s from nose infections?

We have known for years that our healthy gut bacteria is essential to prevent almost every disease, and now research is looking at the rhinosinal microbiome, the healthy bacteria in our nose.
This is now becoming known as Inhalational Alzheimer’s.
The nose is the most direct route to the brain, and bad bacteria in the mucous lining of the airways can damage the brain.
Pathologists now believe there are unknown pathogens in the rhinencephalon, the “nose-smell” (olfacation) system.
Many Alzheimer’s patients start losing their sense of smell as one of the early signs of the disease, and this is probably why.
I am confident that my nasal bacteria is back to normal after having very bad allergies and taking antihistamines from when I was about 16 to when I was 63.
Allergies stopped when the bad diet stopped.

Dr. Susan Lynch at UCSF has found that the nose problem is not so much an unknown pathogen, but a lack of microbial diversity.
Beneficial microorganisms in the nose protect against many pathogens, and one of the best seems to be Lactobacillus sakei, used to make sake and kimchi.
This could explain why Japanese people have comparatively low rates of Alzheimer’s, although rates are rising in Japan because of the Western influence, with meat and dairy replacing rice as a staple food.
When Japanese people migrate to Western countries and adopt a Western diet, they have the same risk as anyone else.
So for the Japanese, it is not a genetic problem, but a diet problem, and this applies to everyone.

AGEs – Advanced Glycation End products

AGEs are formed when food cooked at high temperatures (over 120 degrees C) combines with sugar. AGEs are very damaging to the body, accelerating the ageing process and chronic disease.
AGEs worsen diabetes, kidney disease, Alzheimer’s, inflammation, atherosclerosis (stiffening of the arteries), cardiovascular disease and stroke.
AGEs cause glycation of LDL cholesterol, promoting oxidation, and oxidized LDL is a major factor in atherosclerosis.
AGEs form photosensitizers in the eye lens, leading to cataract development.

To reduce AGEs, never cook at high temperatures (steaming is best, always at 100 degrees C), eat plenty of raw food (salads, and small amounts of fruit), and eliminate all sugar and processed foods.

Conventional Drugs

Drug companies have been trying for years to get rid of Amyloid plaques, thinking they are the cause of Alzheimer’s.
However, the body needs amyloid to protect the brain, so we need to look at what is causing the plaques instead of trying to get rid of them. Latest research shows that Amyloid plaques are antimicrobial, so can be both damaging and protecting!

 

Alzheimer’s – “Diabetes Type 3”

Some researchers are now labeling Alzheimer’s as “Diabetes Type 3” because sugar causes Alzheimer’s.
Sugar also causes diabetes, cardiovascular disease, obesity and many more diseases, mainly due to processed foods.
As with diabetes, where sugar causes insulin resistance, we have insulin resistance in the brain, causing degeneration.
When the brain becomes insulin resistant, it means that glucose cannot enter the brain cells, so those cells die.
However, all is not lost. If we switch to a Ketonegic diet, we can feed our brain with fat instead of sugar. More on this diet below.

Diagnosing the type of Alzheimer’s

Unlike cancer, where we can biopsy a tumour, we must look at historical, biochemical, genetic, imaging, and function information to determine the type of Alzheimer’s.
Of course this rarely happens except in research applications. The doctor simply says the patient has Alzheimer’s and may give a drug which in the long term will not make much difference.
This is a shame, because about half of all cases can be halted, and in some cases substantially improved, by reverting to the correct diet.
Even better would be to eat a correct diet from birth, reducing the risk of Alzheimer’s to near zero, as well as preventing cancer, heart disease, diabetes and other modern diseases.

Exercise

Physical exercise is extremely important to keep the brain and body healthy.
Researchers are not sure why, but LeanMachine says it is obvious:
Exercise burns off the high glucose levels that cause “Diabetes of the Brain” and exercise boosts oxygen levels and circulation in the brain.
Any type of exercise is beneficial, such as:

  • Walking, jogging or running
  • Calisthenics
  • Squats
  • Push-ups, chin-ups
  • Skipping
  • Gardening

Exercises have the added benefit of socialisation in a group, such as:

  • Join a gym
  • Tai-Chi or Yoga classes
  • Athletics clubs
  • Dancing classes

Exercising the Brain

The body has a disturbing property: Anything not used for a while gets broken down to be used somewhere else.
If we do not use a muscle for a week, the body starts breaking it down.
But if we exercise regularly, we stop muscles wasting, and we actually build up our muscles.
If we do not use parts of the brain, the body starts breaking it down.
But if we exercise our brain, we can hang on to the parts we use, and develop new pathways to replace parts we have lost. Exercises such as:

  • Learning a new language
  • Playing a musical instrument
  • Crossword or other puzzles
  • Socialising in groups or clubs

Meditation

Meditation is not normally seen as exercise for the brain, but sitting in a quiet, dark room away from all daily distractions not only promotes a calming effect, but increases various brain-saving hormones.
Meditation, like dreaming, helps the brain sort out the junk memories and recent problems by concentrating on things that have made us feel good in the past.
We may have pleasant memories like sitting on a sandy beach listening to the waves rolling in on a beautiful sunny day. By concentrating on peaceful and pleasant memories, we forget problems with out hectic daily life.

Supplements

The modern diet is lacking in vitamins, minerals, amino acids and other nutrients, mainly because of:

  • Over-farming – growing the same food in the same ground year after year, depleting these vital elements
  • Over-processing – hydrogenation, adding sugar, adding chemicals, overheating
  • Toxins from farming chemicals contaminates the environment
  • Water is contaminated by fluoride and chlorine

The supplements everyone over 50 should take are:
Organic Coconut Oil, taken several times a day, a tablespoon at a time.
LeanMachine considers this one of the best prevention and treatment methods available for Alzheimer’s.
This encourages the body to burn healthy fats instead of sugar, called the Ketogenic Diet which burns ketones, which is what our ancestors did in their natural low-carb diets. See the Ketogenic Diet below.
Coconut oil appears to break down the amyloid plaque buildup in the brain. Perhaps the plaques are no longer required when the brain is fed by healthy fats instead of glucose.
Coconut oil is also the absolute best for cooking, replacing any other fat, because coconut oil remains stable at high temperatures, and is full of MCT (Medium Chain Triglycerides) which go straight to the liver to be burned as fuel, and cannot be stored as fat in the body.
Coconut oil also contains Lauric Acid, which keeps our skin wrinkle-free and healthy.

PS (Phosphatidylserene) is a component of the cerebral cortex’s neuronal membrane, and can improve memory and mood, reduce stress, improve learning and more.
It does this by controlling input and production of choline, acetylcholine, norepinephrine, dopamine and glucose.

NAC (N-Acetyl Cysteine) which helps the body make Glutathione, the body’s natural “Master Antioxidant” that fights cancer, Alzheimer’s and many other conditions.

Curcumin is the active ingredient in Turmeric which has been used for thousands of years for dementia, cancer and many other conditions.

Vitamin B-12 because as we age, our stomach acid levels drop, preventing the high-acid conditions required for B-12 absorption from food. Even more essential for vegans and vegetarians as B-12 mainly comes from animal products.

B-group vitamins because these are vitally important for nerves and brain health.

ALA (Alpha Lipoic Acid) as an antioxidant to help remove heavy metals from the brain, reduce inflammation, and improve the effectiveness of votamins C and E.

Vitamin D3 because over half the ageing population are taking statin medication (which they should NOT) and statins halt production of 7-dehydrocholesterol, the first step in the manufacture of vitamin D3. Worse, many of these seniors are in Aged Care facilities and never see the light of day, so cannot make vitamin D3 from sunlight. If they are ever taken outside, it is only early morning or late afternoon when they cannot get vitamin D3 anyway. More info in my Vitamin D3 article.

Ginkgo Biloba is highly recommended to improve blood flow in the brain. Should not be used in conjunction with prescription blood thinners.

TMG (Trimethylglycine) is an effective methyl donor for the facilitation of methylation processes. Supports a healthy homocysteine level, which in turn supports healthy cardiovascular function and helps prevent Alzheimer’s. Homocysteine, a damaging amino acid, with the aid of TMG, is turned into methionine, a safe and beneficial amino acid. Methylation is essential for DNA repair and production of SAMe, which helps joints, lifts mood, fights depression and protects brain cells from amyloid plaques. Read more in my TMG article.

SAMe (S-Adenosyl Methionine) can help protect the brain and also help treat depression, anger, anxiety which are common symptoms in some Alzheimer’s patients.

Vinpocetine has shown mixed results but mostly beneficial in limited human trials using 10mg 3 times daily.

Vitamin E is recommended to improve the healthy fats in the brain and increase antioxidants.

Benfotiamine with Leucine can help remove glucose and improve insulin resistance.

Many other supplements can help, including:

In addition, many supplements primarily used to treat diabetes will also help prevent Alzheimer’s.

The Cholesterol Fraud

Previous research indicated that high cholesterol was a risk factor for Alzheimer’s.
Again, this was wrong. Doctors started prescribing statin drugs for those people with high cholesterol, or those with signs of dementia with normal cholesterol.
What happened? They got Alzheimer’s WORSE and got it FASTER than patients who did NOT take statins.
Researchers only looked at total cholesterol which is a complete waste of time.

25% of the cholesterol in the body is in the brain, mainly in the myelin sheath.
Around 60% of our brain is fat, mainly in the form of cholesterol.
The myelin sheath (oligodendroglia) that surrounds and protects our neurons are 70% cholesterol, 30% protein.
Starve the brain of healthy fat, and we get Alzheimer’s. Almost guaranteed.
Reduce cholesterol and what happens? The protective myelin sheaths break down as they are starved of cholesterol, allowing the brain cells to be damaged. Damage them enough, and they die. Then we have dementia. Damage enough cells, and the brain can no longer support our basic functions, like breathing. Then we die.
This is why statin drugs are BAD.
Sure, in some cases, they can slightly reduce risk of heart attacks, but they INCREASE death from all other causes, including Alzheimer’s.
The net result is that on average, we will not live a day longer on statin medication.
Statins will give us lousy final years with muscle breakdown, osteoporosis, more sickness and dementia.
We need plenty of healthy fats like coconut oil, walnuts, avocados, fish, eggs, butter from grass-fed cows, unheated olive oil.
We must NOT consume bad fats: Canola oil, margarine, anything hydrogenated, anything heated over 120 degrees C.
Cholesterol is NOT the enemy.
We NEED cholesterol, especially HDL (High Density Lipoprotein) cholesterol which reduces inflammation, and helps clean up the body (like a garbage collector). Without HDL Cholesterol, we die within 24 hours.
We also need LDL (Low Density Lipoprotein), still incorrectly called “bad” cholesterol, as we die without it.
LDL has antimicrobial effects, so the idea that we should drive it down to zero is ludicrous. LDL is essential to transport nutrients around the body (and into the brain) as well as helping the body manufacture hormones and other important products. LDL was essential for our evolutionary ancestors millions of years ago, and we still need it.
The brain is mostly fat, and 40% of the brain is CHOLESTEROL.
Many things that were protective in our native environment are problems in our modern environment, but if we go back to our ancestral diet, problems are resolved.
Studies show time after time that people with low cholesterol die young, while people with normal to high cholesterol live longest.
These studies are ignored by the big drug companies. Because statin sales make them billions of dollars, of course they continue the Big Cholesterol Lie, one of the biggest frauds in medical history. Their own study showed increased deaths and terrible side effects so they stopped the study short at that time, supposedly to “save patient’s lives” when the opposite was true.

The dangerous cholesterol is VLDL (Very Low Density Lipoprotein) which cannot easily be tested.
Because triglycerides contain some VLDL, labs estimate VLDL value by simply taking a percentage of triglycerides.
High triglycerides are much more of a danger signal than high cholesterol, and are almost always related to obesity, poor diet of processed foods, especially dangerous fats.

The Big Fat Lie

We have been told for decades that fat is bad for us.
Forget about “low fat” or “fat free” diets.
Another big fat lie, coming from a scientist who plucked figures out of a study to suit an argument he was proposing.
When the data was analysed completely, many decades later, it showed the complete opposite.
The largest and longest study in the world was the Framingham study which showed that those who ate the most fat lived longer than those who ate the least.
Fat is not unhealthy in general, in fact it is essential for health.
The UNHEALTHY fats are man-made artificial fats (margarine, Canola oil) and other processed fats that are hydrogenated to improve shelf life and heated to extremes during manufacture, often going rancid in the process, causing oxidised VLDL (Very Low Density Lipoprotein), the REAL dangerous “food”.
What is REALLY bad is carbohydrates, and when manufacturers remove fats from food, they replace them with carbohydrates, causing most “modern” diseases including Alzheimer’s and Diabetes.

The Ketogenic Diet

For the first two million years of human life on Earth, carbohydrate consumption was very low.
Carbohydrates were uncommon, with the majority of food being nuts, seeds, eggs, fish, fruit and vegetables. Meat was eaten very rarely when an animal was killed.
These people did not burn carbohydrates for energy, they burned FAT. In particular, ketones, the basis of the ketogenic diet.
A ketogenic diet means maintaining a fasting state of ketosis. Ketones are produced when the body is in a state of ketosis.
Ketones fuel cells using a different pathway from glucose.
Glucose has to have insulin to allow glucose into cells, but as we all should know, our typical modern diet is loaded with carbohydrates, forcing the pancreas into overdrive making enough insulin.
Eventually our cells become insulin resistant, so the pancreas produces even more insulin to force glucose into the cells, creating even more insulin resistance.
We are now a full-blown diabetic, and when the pancreas starts shutting down, we need insulin injections for the rest of our life.
However, when we feed the cells with ketones, they simply enter the cell naturally, and do NOT require insulin or anything else to do so.
This is critically important for five of our modern diseases: Obesity, Cancer, Diabetes, Cardiovascular and Alzheimer’s, all caused or aggravated by high blood glucose, bad fats and inflammation.
Ketones are also signaling molecules as well.

Benefits of the ketogenic diet include:

  • Helps the body express new restorative and healing genes
  • Reduces inflammation (underlying cause of nearly every disease)
  • Stimulates the immune system
  • Aids weight loss
  • Stops or slows degenerative disease
  • Reduces risk of Alzheimer’s, Cancer, Cardiovascular, Diabetes and Obesity

The Anti-Alzheimer’s diet

Spices

Add these spices to every meal possible.
Of course they will spice up any meal, but also help clear the brain of problems and reduce risk of cardiovascular disease, cancer, diabetes and many more modern illnesses.

  • Sage – one of the best brain-saving spices
  • Cloves – one of the most potent antioxidants
  • Curry – a blend of other great spices
  • Ginger – reduces inflammation and improves immunity
  • Turmeric – for colour, flavour and Curcumin
  • Ceylon Cinnamon – Better and safer than regular cinnamon

Ketogenic Diet – Healthy fats, intermittent fasting.
Read How Cyclical Ketosis can help combat Chronic Fatigue

Avoid Trans Fats
Read Trans Fats Linked to Increased Risk for Alzheimers

Avoid Processed Foods
Only shop in the greengrocer department at the Supermarket, preferably the organic section. Buy or grow your own real food. Nothing in a bag, box, tin because toxic ingredients are sure to be added.

Avoid AGEs
Forget fried foods. Steaming is the best way to cook. Never Microwave. Eat raw salads daily.


This section often updated. Please come back soon (if you remember!)

LeanMachine online supplements

Updated 20th January 2020, Copyright © 1999-2020 Brenton Wight and BJ&HJ Wight trading as Lean Machine abn 55293601285

76 Evidence-Based Health Benefits of Noni

© 3rd January 2020 GreenMedInfo LLC. This work is reproduced and distributed with the permission of GreenMedInfo LLC. Want to learn more from GreenMedInfo? Sign up for the newsletter here www.greenmedinfo.com/greenmed/newsletter
Reproduced from original article:
https://www.greenmedinfo.com/blog/76-evidence-based-health-benefits-noni
Posted on: Friday, January 3rd 2020 at 4:15 pm

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of GreenMedInfo or its staff.

Fibromyalgia

What is Fibromyalgia?

Fibromyalgia is a chronic condition, typically very painful, especially in response to pressure, and sometimes patients have symptoms like stiff muscles, joints and connective tissues.
Other symptoms often include depression, anxiety, sleep disturbance, difficulty swallowing, bowel and bladder problems, numbness and tingling, muscle spasms or twitching, weakness, nerve pain, palpitations,
cognitive dysfunction (“foggy thinking”).
Around 2% of the population are affected, usually between the ages of 20 and 50, although not all patients have all symptoms.
Women are nine times more likely than men to suffer from the condition, giving weight to the theory that hormones play a big part in the cause and treatment.
Diagnosis is difficult because there is no formal test. Symptoms are vague and similar to many other conditions.
Often patients with celiac disease are mistakenly diagnosed with Fibromyalgia, and do better on a gluten-free diet.
In fact, nearly everyone will do better on a gluten-free diet, or even better, a diet free from all grains, flour and any other product of grains, regardless of refined, wholemeal or any other form.
Some medical specialists say it is “all in the head” but few patients would agree with this!

Testing

Although there is no formal testing for fibromyalgia, the following tests should be arranged by the doctor to eliminate some factors that may indicate or aggravate Fibromyalgia:

  • Ferritin (Iron Study) – A serum ferritin level under 50 ng/ml means a 650% increased risk for Fibromyalgia
  • Thyroid Function – If autoimmune hypothyroidism is present, it should be treated first to see if Fibromyalgia symptoms subside
  • Other autoimmune conditions – Lupus, Rheumatoid Arthritis and others can resemble Fibromyalgia symptoms and should be treated first
  • CRP (C-Reactive Protein) – An inflammation marker. Source of any inflammation should be treated first
  • The FM/a blood test (plasma and PBMC (Peripheral Blood Mononuclear Cells) – Tests cytokine concentration. Low cytokines may indicate Fibromyalgia

Treatment

Doctors say there is no known cause or cure. However, some approaches can be very effective in reducing symptoms, including:

Therapeutic options

  • Mindfulness Training reduces psychological distress and depression
  • Yoga, Tai-Chi and other stretching exercises are helpful as they stimulate the lymph glands, increasing our HDL (good cholesterol), improving waste product and toxin removal, also reducing pain, fatigue, mood, cortisol levels and improves coping ability

Diet

  • Raw Food has been shown in studies to significantly improve the majority of fibromyalgia patients
  • Vitamin C and Broccoli consumption in a study found that the combination of 100mg of vitamin C from food, plus a 400mg broccoli supplement reduced pain by 20% and decreased 17% in Fibromyalgia impact scores

Things to avoid

Exposures to toxins definitely increase fibromyalgia risk:

  • Breast Implants have been linked to cancer, autoimmune disease, fibromyalgia and chronic pain
  • Aspartame (an artificial sweetener) should be eliminated from the diet, as it turns into formaldehyde in the body, which can aggravate fibromyalgia.
    Natural sweeteners such as Erythritol, Xylitol and pure Stevia are healthy alternatives
  • MSG (MonoSodium Glutamate) should be eliminated from the diet. Known to cause headaches and fibromyalgia
  • Vaccine Adjuvants containing mercury or aluminium have been shown to cause musculoskeletal pain conditions like fibromyalgia
  • Fluoride comes from fluoridated tap water, foods irrigated with fluoridated water, toothpaste, dental treatments and antibiotics, and must be avoided. A fluoridated water supply should be switched to rainwater and/or install a Reverse Osmosis water system for all drinking and cooking. Ordinary water filters do not remove fluoride, and even boiling water makes little difference

Prescription Medications increase risk

Many prescription medications increase risk of fibromyalgia, or actually cause it.

  • Statin Drugs reduce CoQ10 and vitamin D3, causing hundreds of health problems, including fibromyalgia and muscle pain, vastly outweighing any benefit in many cases
  • Prescription antidepressants like Celexa (Citalopram), Paxil (Paroxetine) and Prozac (Fluoxetine) include fluoride which makes fibromyalgia even worse, and causes weight gain.
    Antidepressants increase risk of cancer by over 40%, and most of the time do not work any better than a placebo
  • Many drugs contain bromide, which is even worse than fluoride, and more easily displaces iodine from the thyroid gland
  • Antibiotics destroy many bad bacteria, but also much of the good bacteria as well, compromising our immune system, which can take up to two years to rebuild
  • Paracetamol, Panadol, Tylenol and other names for acetaminophen should be avoided as studies show them to start causing liver issues even at the recommended dose two 500 mg tablets four times a day (4000 mg) for a few days. Unfortunately, patients who experience a lot of pain invariably over-dose, and just a 50% increase starts causing severe liver damage. The advertising slogan “safe and effective” is one of the biggest lies of the drug industry, and the most common cause of liver poisoning in the Western world. The majority of all patients on the liver transplant waiting list are there because of Panadol overdose. Panadol also reacts with an enzyme in the body to destroy our natural glutathione, which is one of the body’s main defenses against pathogens, often called the “master antioxidant”. Less glutathione means more Fibromyalgia

Here is a list of some drugs commonly prescribed that contain Fluoride or Bromide, two halogens that displace iodine from the thyroid and cause hypothyroidism, Hashimoto’s disease, depression, weight gain, hair loss, cancer, and will aggravate Fibromyalgia:

  • Advair (fluticasone) – fluoride
  • Alphagen (brimonidine) – bromide
  • Atrovent (Ipratropium) – bromide
  • Avelox (moxifloxacin) – fluoride
  • Adovart (dulasteride) – fluoride
  • Celebrex (celecoxib) – fluoride
  • Celexa (citalopram) – fluoride and bromide
  • Cipro (ciprofloxacin) – fluoride
  • Clinoril (sulindac) – fluoride
  • Combivent (from the ipratropium) – bromide
  • Crestor (rosuvastatin) – fluoride
  • Diflucan (fluconazole) – fluoride
  • DuoNeb (nebulized Combivent) – fluoride
  • Enablex (darifenacin) – bromide
  • Flonase (fluticasone) – fluoride
  • Flovent (fluticasone) – fluoride
  • Guaifenex DM (dextromethorphan) – bromide
  • Lescol (fluvastatin) – fluoride
  • Levaquin (levofloxacin) – fluoride
  • Lexapro (escitalopram) – fluoride
  • Lipitor (atorvastatin) – fluoride
  • Lotrisone topical cream – fluoride
  • Paxil (paroxetine) – fluoride
  • Prevacid (lansoprazole) – fluoride
  • Protonix (pantoprazole) – fluoride
  • Prozac (fluoxetine) – fluoride
  • Pulmicort (budesonide) – fluoride
  • Razadyne (galantamine) – bromide
  • Risperdal (risperidone) – fluoride
  • Spiriva (tiotropium) – bromide
  • Tobra Dex (from dexamethasone) – fluoride
  • Travatan (travoprost) – fluoride
  • Triamcinolone – fluoride
  • Vigamox (moxifloxacin) – fluoride
  • Vytorin (from eztimibe) – fluoride
  • Zetia (eztimibe) – fluoride

An immune response to intestinal bacteria may cause some symptoms, so an alkaline diet with plenty of enzyme-rich raw vegetables and fresh fruit may help, along with a little cheese, yogurt, whey, fermented vegetables such as Sauerkraut, and/or supplemental probiotics such as Acidophilus
to build up beneficial intestinal bacteria. 75% of our immune system is in the gut, and this is where the immune system often first breaks down.

MSG (monosodium glutamate) has been shown to aggravate symptoms, so most processed food, which contains MSG, often hidden in the ingredients list by being called other names or chemicals, should be eliminated.

Eliminating yeast from the diet may also help. Yeast is a raising agent found in most breads and other flour-based baked foods, also Vegemite. Changing to a fresh food diet of vegetables and fruit can eliminate yeast, lose excess weight, build immunity and improve general health.

Casein from milk and other milk products may also help, although some people are sensitive to dairy products and do better with no milk or other dairy products.

Food allergies can be a problem and I would start by eliminating wheat, flour, bread, cakes, anything made from flour, sugar, soy, milk, corn, eggs and nuts for at least a week or two.
If that helps, introduce them back into the diet one at a time (except sugar, which should be omitted forever, and all flour products), until the culprit is found.

If that is not enough, see my Vaccinations article and read about the relationship between Panadol, Vaccinations, Glutathione and Autism.

Many Fibromyalgia patients also suffer from IBS (Irritable Bowel Syndrome), CFS (Chronic Fatigue Syndrome), RA (Rheumatoid Arthritis) and SLE or Lupus (Systemic Lupus Erythematosus), but the above treatments can improve all of these conditions.
While these natural alternatives may not work for everyone, nearly all patients report improvement in their condition, and of course, these are all good for weight loss, fighting diabetes, cardiovascular disease, Alzheimer’s disease, better sleep, improved mood, reduced pain, better pain tolerance, building muscle and reduced cancer risk. Many patients are deficient in GH (growth hormone) so high-intensity exercise and weight loss will help by increasing natural production of Growth Hormone.

LeanMachine online supplements

Updated 24th January 2020, Copyright © 1999-2020 Brenton Wight and BJ&HJ Wight trading as Lean Machine abn 55293601285

Excess Body Fat Can Age Your Brain Faster Than Muscle


Reproduced from original article:
https://articles.mercola.com/sites/articles/archive/2020/01/09/obesity-and-brain-health.aspx
Analysis by Dr. Joseph Mercola Fact Checked image
January 09, 2020

excess body fat and brain health

STORY AT-A-GLANCE

  • Increasing research shows that maintaining healthy levels of body fat and greater muscle mass has an effect on your brain health and may slow your rate of cognitive aging
  • People with higher amounts of abdominal fat had worse fluid intelligence with age, while those with greater muscle mass were more protected against such declines
  • Women who had greater muscle mass tended to have better scores in fluid intelligence during the study period
  • Past research has linked midlife obesity with an increased risk of mild cognitive impairment, changes in short-term memory and executive functioning and dementia
  • In addition to regular exercise to increase muscle mass, eating a ketogenic diet to maintain a healthy body weight and avoid obesity may support your brain health as you age

Staying fit as you age is about far more than aesthetics. Increasing research shows that maintaining healthy levels of body fat and greater muscle mass has an effect on your brain health and even your rate of cognitive aging. It’s known, for instance, that being obese in midlife and early late-life is associated with worse cognitive aging.1

What’s more, the amount of muscle and fat you have may be a more important factor in how your level of fluid intelligence decreases over time than your chronological age. Your chronological age, i.e., your age in years, is just a numerical measurement, but your real age is your biological age as dictated by your choices and habits, as well as your modifiable risk factors like levels of muscle and fat.

While many people tend to gain fat and lose muscle mass as they age, this can be largely combated by staying active and eating right — lifestyle choices that will influence your cognitive function significantly.

More Muscle, Less Fat Protects Your Brain

In a study by Iowa State researchers, data from 4,431 adults were examined to compare levels of lean muscle mass, abdominal fat and subcutaneous fat with changes in fluid intelligence — the ability to solve problems in new situations — over a six-year period.2,3

Those with higher amounts of abdominal fat had worse fluid intelligence with age, while those with greater muscle mass were more protected against such declines. In fact, women who had greater muscle mass tended to have better scores in fluid intelligence during the study period.

Study co-author Auriel Willette, assistant professor of food science and human nutrition at Iowa State University, said in a news release, “Chronological age doesn’t seem to be a factor in fluid intelligence decreasing over time. It appears to be biological age, which here is the amount of fat and muscle.”4

What’s more, the study revealed a link between the immune system and how changes in fat levels affect cognition. Previous research suggests a higher body mass index (BMI) leads to greater immune system activity in the blood, which in turn activates the immune system in the brain, with a negative outcome on cognitive function.5

The featured study also found that changes in white blood cells called lymphocytes and eosinophils explained the link between abdominal fat and worsening fluid intelligence in women. In men, basophils, another type of white blood cell, were linked to about half of the link between fat levels and fluid intelligence, the study found.6

“Lymphocytes, eosinophils, and basophils may link adiposity to cognitive outcomes,” the researchers explained.7 Similar research has revealed that overweight and obese individual have greater brain atrophy in middle-age, corresponding with an increase in brain age of 10 years.8

How Obesity Affects Your Brain

Obesity has multiple effects on the brain, including anatomically speaking. Obese individuals may have reduced gray matter in brain regions such as the hippocampus, prefrontal cortex and other subcortical regions. Atrophy in the hippocampus, in turn, has been linked to Alzheimer’s disease.9

Gray matter is the outer layer of the brain associated with high-level brain functions such as problem-solving, language, memory, personality, planning and judgment. Even in elderly people who are otherwise cognitively normal, obesity is associated with measureable deficits in brain volume in the frontal lobes, anterior cingulate gyrus, hippocampus, and thalamus compared to individuals with a normal weight.10

Further research published in Radiology found that obesity may lead to alterations in brain structure, shrinking certain regions.11 Among men, higher total body fat percentage was linked to lower brain gray matter volume. Specifically, 5.5% greater total body fat percentage was associated with 3,162 mm3 lower gray matter volume.

Among men, 5.5% greater total body fat was also associated with 27 mm3 smaller globus pallidus volume, an association also seen in women. In women, 6.6% greater total body fat percentage was associated with 11.2 mm3 smaller globus pallidus volume.

The globus pallidus is a brain region that plays a role in supporting a range of functions, including motivation, cognition and action.12 Obesity was also associated with changes in white matter microstructure, which may be related to cognitive function.13

Cognitively speaking, there’s also a strong link between obesity and deterioration in cognitive function, as well as to other brain disorders such as dementia, anxiety and depression. Further, past research has linked midlife obesity with an increased risk of mild cognitive impairment, changes in short-term memory and executive functioning and dementia.14

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Obesity-Associated Health Problems Also Harm Your Brain

Obesity’s effects on brain health are also due to its associated health problems, including heart disease, diabetes and atherosclerosis, each of which can have its own deleterious effects on your brain. For instance, as noted in Frontiers in Neuroscience:15

“Obesity-derived vascular problems, such as atherosclerosis and arteriosclerosis, which are systemic diseases, are known to affect the steady blood flow of vessels that feed the brain, thus contributing to cognitive impairment or even stroke, where large areas of the brain die due to the stop in the blood flow of a major brain artery caused by a blood clot.”

In terms of diabetes, of which obesity is a key risk factor, having this condition in midlife is associated with a 19% greater cognitive decline over 20 years compared with not having the condition.16 Even those with prediabetes had significantly greater cognitive decline than those without.

Indeed, “Epidemiological studies have linked type-2 diabetes mellitus with cognitive impairment and dementia, with insulin resistance and hyperglycemia as the probable mechanistic links,” researchers noted.17

Coming full circle, eating a highly processed, junk food diet not only increases obesity risk but also can lead to normal but elevated blood sugar levels that, in turn, can lead to impaired glucose metabolism and Type 2 diabetes. Both diabetes and higher fasting glucose levels are linked with lower total brain volume.18

Impaired glucose metabolism is then associated with neurodegeneration that impairs cognitive function. This connection begins not in old age but much earlier, such that following a healthy lifestyle in young adulthood may be protective against cognitive decline later.19

The Inflammation Connection

Obesity can trigger chronic inflammation in your body, and chronic inflammation in your brain (neuroinflammation) is known to impair neurogenesis, your brain’s ability to adapt and grow new brain cells. It’s also linked to neurodegenerative disorders such as Alzheimer’s disease (AD), and it’s been suggested that “Obesity may serve as an amplifier or initiator of the chronic inflammation observed in AD patients.”20

Further, higher levels of inflammatory markers have also been associated with lower brain volume, including “greater atrophy than expected for age.”21 Excess body fat, particularly visceral fat, is also related to the release of proteins and hormones that can cause inflammation, which in turn can damage arteries and enter your liver, affecting how your body breaks down sugars and fats.

According to a study in the Annals of Neurology, “[A]dipose-tissue derived hormones, such as adiponectin, leptin, resistin or ghrelin, could also play a role in the relation between adipose tissue and brain atrophy.”22 Further, obesity may also be associated with lower volume in brain regions that regulate food-reward circuitry,23 possibly influencing overeating.

Strength Training Is Good for Your Brain

While obesity takes a toll on your brain, increased muscle mass protects it, which is likely one reason why strength training has been found to be beneficial for your brain. In other words, your body’s physical strength may serve as a marker of your brain power.

In fact, strength training is known to trigger beneficial neurobiological processes,24 leading to positive functional brain changes, including in the frontal lobe, with corresponding improvements in executive functions. One systematic review even found that strength training led to less white matter atrophy in the brain, with researchers noting:25

“Taken together, during aging processes, a substantial decline in muscular strength, especially in lower limb muscles, occurs, and accumulating evidence suggests that lower muscular strengths are linked to poorer cognitive performance.

Hence, resistance (strength) exercises (a single bout of resistance exercise, also referred to as acute exercise) and resistance (strength) training (more than one resistance exercise session, also referred to as chronic exercise … ) seem to be promising activities to ensure the preservation of physical functioning and cognitive functions with aging.”

Regular strength training, in addition to other forms of exercise and daily activity, is an important strategy for keeping your brain sharp and may help to offset some of the cognitive decline that occurs with age.

Avoid Obesity and Protect Your Brain With a Ketogenic Diet

While obesity may accelerate neurodegeneration, regular exercise to increase your muscle mass will be protective. Further, eating a ketogenic diet will help protect your brain from free radical damage and will supply the cells with preferred fuel while also helping you to lose weight and avoid obesity.

A ketogenic diet is high in healthy fats and low in net carbohydrates (total carbs minus fiber), prompting your body to start burning fat as its primary fuel, rather than sugar. This produces ketones, which not only burn efficiently but are also a superior fuel for your brain. Ketones also generate fewer reactive oxygen species (ROS) and less free-radical damage.

One of the simple strategies you can implement is to take ketone precursors like refined MCT oils of caprylic acid (C-8). The eight-chain carbon fats are readily converted to ketones. I personally use up to 5 ounces of our Ketone Energy when I have maxed out my protein and carb intake and need a source of healthy clean fat. This keeps my ketone level around 1 to 2.0 mmol/l. Just recognize that you have to build up to a high dose of MCT oil slowly or you will have problems with loose stools.

Recent studies have also demonstrated the benefits of nutritional ketosis for brain health. In one, researchers found a ketogenic diet improved neurovascular function, in part by improving your gut microbiome.26

In a second study, the researchers concluded a ketogenic diet acted as a veritable “fountain of youth” in their animal study by significantly improving neurovascular and metabolic functions, compared to the animals eating an unrestricted diet.27 Releasing ketones into your bloodstream helps preserve brain function and protects against cognitive impairment and other neurodegenerative diseases.28

KetoFasting, the program I developed and detail in my book, “KetoFast: A Step-By-Step Guide to Timing Your Ketogenic Meals,” combines a cyclical ketogenic diet and intermittent fasting with cyclical partial fasting to optimize health and longevity.

Not only can KetoFasting help you to lose weight, but your cognition typically improves thanks to the biological cleansing and regeneration that occurs throughout your body, including your brain.

Sources and References